Development of high fat diet-induced obesity and leptin resistance in C57Bl/6J mice

By S. Lin, T. Thomas, L. Storlien, XF Huang. International Journal of Obesity, 2000

607 citations recorded by the source corpus.

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

Orexin activation counteracts decreases in nonexercise activity thermogenesis (NEAT) caused by high-fat diet. (2017, background): “EE that occurred prior to long-term differences in food intake, which has been shown following long term exposure to HFD [33].”
Time restricted feeding without reducing caloric intake prevents metabolic diseases in mice fed a high fat diet (2012, unknown): “Voluntary energy intake is often independent of dietary fat content both in rodents and in human twins (examples include but are not limited to Bray et al., 2010; Fujisaka et al., 2011; Hosooka et al., 2008; Kennedy et al., 2007; Lin et al., 2000; Saltzman et al., 1997; Samuel et al., 2004), and the”
High-fat diet-induced obesity in animal models (2010, background): “The researchers suggested that early in high-fat feeding, animals are sensitive to the food-lowering effect of leptin but despite the reduction in food intake animals become fat as a result of the increase in food efficiency, leading to an increase in plasma leptin levels that is followed by insensi”