Rapid actions of plasma membrane estrogen receptors.

By M. Kelly, E. Levin. Trends in endocrinology and metabolism, 2001

683 citations recorded by the source corpus.

Subfields: Endocrinology, Diabetes and Metabolism, Genetics, Molecular Biology

View Paper: https://www.semanticscholar.org/paper/b93fe33d388dc1df3f7e05244d5e5bb21d8f25df

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Endocrinology, Diabetes and Metabolism

1. R. Simerly
2. Bart Staels
3. Bruce S. McEwen
4. Christine Marcelle Heim
5. Elisabeth B. Binder

In Genetics

1. R. Simerly
2. Mitchell Dowsett
3. Bruce S. McEwen
4. Christine Marcelle Heim
5. Elisabeth B. Binder

In Molecular Biology

1. Bart Staels
2. Bruce S. McEwen
3. R. Simerly

Citations in the corpus

• Overview of Nomenclature of Nuclear Receptors (2006, background): “Another type of NR cross-talk, which has been recognized only recently, is the so-called “nongenomic” actions of several receptors that induce very rapid cellular effects (Wehling, 1997; Picard, 1998; Valverde et al., 1999; Kelly and Levin, 2001; Cato et al., 2002).”
• Sex, stress and the hippocampus: allostasis, allostatic load and the aging process. (2002, background): “…potent as 17␣-estradiol vs. oxidative damage Genomic actions: 17␤-estradiol is more potent; anti-estrogen blockade For detailed summaries, see [79] [80] [97] [124]. include substances that are estrogenic [90] and estrogenic substances have neuroprotective effects against free-radical damage which…”
• Wired for reproduction: organization and development of sexually dimorphic circuits in the mammalian forebrain. (2002, background): “…SIMERLY In addition to the direct genomic actions of nuclear hormone receptors, steroid hormones can regulate transcriptional events by altering expression of other transcription factors such as the protooncogene Fos (Insel 1990) or by coupling to second-messenger pathways (Kelly & Levin 2001).”
• Mechanisms of tamoxifen resistance. (2004, background): “Oestrogen-activated membrane ER a has been shown to activate the insulin-like growth factor-1 receptor (IGF-1R) cell signalling cascade by direct binding to IGF-1R, followed by its phosphorylation, a process that was found to be dependent on mitogen-activated protein kinase (MAPK) kinase activity…”
• Changes in proinflammatory cytokine activity after menopause. (2002, background): “ERs have been detected in many cells that reside in bone tissue (272–278), suggesting that menopause may have direct consequences on cytokine secretion by cells located within the bone microenvironment.”