Tumor‐associated macrophages (TAM) as major players of the cancer‐related inflammation

By G. Solinas, G. Germano, A. Mantovani, P. Allavena. Journal of Leukocyte Biology, 2009

1,318 citations recorded by the source corpus.

View Paper: https://www.semanticscholar.org/paper/b776c9b3e14f3fad0f1d8069c82ed0f3806230ed

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Citations in the corpus

Tumor-Associated Macrophages as Major Players in the Tumor Microenvironment (2014, background): “Certain growth factors including vascular endothelial growth factor (VEGF), endothelin 2, and platelet-derived growth factor (PDGF) have also been reported to promote monocyte/macrophage recruitment [32–34].”
Macrophages in Tumor Microenvironments and the Progression of Tumors (2012, background): “M1 macrophages express a series of proinflammatory cytokines, chemokines, and effector molecules, such as IL-12, IL-23, TNF-α, iNOS and MHCI/II.”
CCL18 from Tumor-Associated Macrophages Promotes Breast Cancer Metastasis via PITPNM3 (2011, neutral): “Among them, tumorassociated macrophages (TAMs) are the most notable migratory hematopoietic cell type (Mantovani and Sica, 2010; Solinas et al., 2009).”
Biomaterial based modulation of macrophage polarization: A review and suggested design principles (2015, background): “This has led to an intense research focus on TAM targeted therapies within the cancer field [66,67].”
The polarization of immune cells in the tumour environment by TGFβ (2010, background): “In addition, the production of CCL22 by TAMs surrounding tumours might mediate T Reg cell trafficking to the tumour bed through CCR4 (Ref”
Targeting the tumour vasculature: insights from physiological angiogenesis (2010, background): “Recruitment of these cells can be mediated by a large number of tumour-derived chemoattractants, most notably colony stimulating factor 1 (CSF1), CCL2, CCL5 and VEG”