Commensal bacteria (normal microflora), mucosal immunity and chronic inflammatory and autoimmune diseases.

By H. Tlaskalova-Hogenova, R. Štěpánková, T. Hudcovic, L. Tučková, B. Cukrowska, R. Lodinová-Žádníková, H. Kozakova, P. Rossmann, J. Bartova, D. Sokol and 7 more. Immunology Letters, 2004

772 citations recorded by the source corpus.

Subfields: Endocrinology, Food Science, Molecular Biology

View Paper: https://www.semanticscholar.org/paper/af8d47fd409e3ffcb5008a9991afd337889f89ac

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Molecular Biology

Citations in the corpus

Commensal Clostridia: leading players in the maintenance of gut homeostasis (2013, background): “Germ-free mouse models, compared to control animals, showed increased mucus accumulation in the cecum, water retention, extended epithelial cell cycles, and decreased peristalsis in the large intestine [54].”
High-throughput clone library analysis of the mucosa-associated microbiota reveals dysbiosis and differences between inflamed and non-inflamed regions of the intestine in inflammatory bowel disease (2011, background): “The microbiota is also, however, a major source of antigens, including lipopolysaccharides, peptidoglycan, lipoproteins, flagellin and unmethylated CpG-containing DNA, all of which can activate both innate and adaptive immune responses [11,12].”
Probiotics and immunity (2009, background): “Pathogen resistance, both by direct interaction with pathogenic bacteria and by influencing the immune system, is influenced greatly by the intestinal microbiota [9, 10].”