Arsenic toxicity and potential mechanisms of action.
By M. Hughes. Toxicology Letters, 2002
1,595 citations recorded by the source corpus.
Subfields: Environmental Chemistry, Health, Toxicology and Mutagenesis, Molecular Biology
View Paper: https://www.semanticscholar.org/paper/a9409a7160fa5783d98e33f93bbaf67e0d7bd907
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Citations in the corpus
- Biomass soaking treatments to reduce potentially undesirable compounds in the edible seaweeds sugar kelp (Saccharina latissima) and winged kelp (Alaria esculenta) and health risk estimation for human consumption (2018, background): “iAs is a known human carcinogen associated with liver, bladder, lung and skin cancers (Hughes 2002).”
- Use of Fish as Bio-indicator of the Effects of Heavy Metals Pollution (2015, background): “Chronic exposures can result in the accumulation of the metalloid to toxic levels and is responsible for several disease conditions [91].”
- Toxic Heavy Metal and Metalloid Accumulation in Crop Plants and Foods. (2016, background): “Whereas methylated As, at least in the pentavalent state, is generally considered less toxic than As(III) and As(V), methylated Hg is more toxic than Hg(II) for most organisms (45, 65).”
- Arsenic: toxicity, oxidative stress and human disease (2011, background): “…one of which concluded that there was no arsenic‐induced increase in NO• generation in hepatocytes and human liver cells, which inhibited inducible NO synthase gene expression in cytokine‐stimulated human liver cells and hepatocytes (Hughes, 2002; Flora et al., 2008; Germolec et al., 1996).”
- Heavy Metals Toxicity and the Environment (2012, neutral): “Arsenic trioxide is methylated to two major metabolites via a non-enzymatic process to monomethylarsonic acid (MMA), which is further methylated enzymatically to dimethyl arsenic acid (DMA) before excretion in the urine [40, 47].”