Dendritic cell regulation of TH1-TH2 development
By M. Moser, K. Murphy. Nature Immunology, 2000
1,291 citations recorded by the source corpus.
Subfields: Immunology
View Paper: https://www.semanticscholar.org/paper/a76e07701a055ee04d965ab3bff14e57d864ba8a
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In Immunology
- Yong‐jun Liu
- R. Steinman
- C. Reis e Sousa
- A. Lanzavecchia
- F. Sallusto
- H. Hemmi
- R. Kastelein
- Jacques Banchereau
Citations in the corpus
- Repetitive Injections of Dendritic Cells Matured with Tumor Necrosis Factor α Induce Antigen-specific Protection of Mice from Autoimmunity (2002, background): “, via CD40-CD154 interaction) leads to full DC maturation, cytokine production, and Th1 or Th2 polarizations (2).”
- The role of oxidative stress in ambient particulate matter-induced lung diseases and its implications in the toxicity of engineered nanoparticles. (2008, background): “Depending on the cytokine milieu and other variables, DC could initiate a primary Th2 response in regional lymph nodes [90–95].”
- Induced recruitment of NK cells to lymph nodes provides IFN-γ for TH1 priming (2004, methodology): “This work was supported by grants from the Swiss National Science Foundation (31-63885) and from the European Commission (‘Memovax’ QLK2-CT-2001-01205 and ‘Main’ FP6-502935).”
- Pathogen-specific T Regulatory 1 Cells Induced in the Respiratory Tract by a Bacterial Molecule that Stimulates Interleukin 10 Production by Dendritic Cells (2002, background): “LPS, dsRNA, and CpG ODN promote maturation of DC1, which direct naive T cells to Th1 subtype (7, 9, 10).”
- IL‐17: prototype member of an emerging cytokine family (2002, background): “Characterization of the cytokine expression profile of individual T cells within the population by cytometric single-cell analysis revealed that the IL-17-expressing cells represent a distinct population from the traditional Th1 profile [34–36] and are characterized by the production of IL-17, GM-CS”
- Regulation of T cell immunity by dendritic cells. (2001, neutral): “Further-(Moser and Murphy, 2000).”
- Inflammatory signals in dendritic cell activation and the induction of adaptive immunity (2009, background): “More importantly, our data argue against the notion that in the absence of polarizing stimuli, newly primed CD4 T cells differentiate to Th1 or Th2 by default (78, 79).”