Interactions between extracellular matrix and growth factors in wound healing

By G. Schultz, A. Wysocki. Wound Repair and Regeneration, 2009

1,067 citations recorded by the source corpus.

Subfields: Biomaterials, Immunology and Allergy, Rehabilitation

View Paper: https://www.semanticscholar.org/paper/a7604f61fc8f12dc1c12cb36d4139beb88f38231

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Biomaterials

1. Rui Yu
2. Hualei Zhang
3. Valerie M. Weaver
4. Baolin Guo

In Immunology and Allergy

1. Valerie M. Weaver
2. Holly N. Wilkinson
3. M. Hardman
4. Pengfei Lu

In Rehabilitation

1. Rui Yu
2. Baolin Guo

Citations in the corpus

• Transition from inflammation to proliferation: a critical step during wound healing (2016, background): “, PDGF, transforming growth factor (TGF)-b and bFGF, produced by platelets and macrophages in the wounds [35, 36].”
• Conductive Biomaterials as Bioactive Wound Dressing for Wound Healing and Skin Tissue Engineering (2021, background): “1007/s40820-021-00751-y © The authors a series of cellular activities for many types of cells, which is crucial in wound healing [64, 271].”
• The extracellular matrix at a glance (2010, background): “In some instances, the release of transforming growth factor (TGF-) by tension and MMPs induces EMT of the resident epithelium (Schultz and Wysocki, 2009; Wipff et al., 2007; Xu et al., 2009).”
• Extracellular Matrix and Dermal Fibroblast Function in the Healing Wound (2014, background): “Since the ECM sequesters and regulates exposure of the fibroblast to soluble cytokines, it is not possible to completely dissociate the role of these signaling molecules from that of the fibroblast’s proteinacious matrix microenvironment.(4) However, this review focuses on the classic, macromolecula”
• The myofibroblast matrix: implications for tissue repair and fibrosis (2013, neutral): “It has been generally understood that elastin production by fibroblasts is low or absent following injury, which partly accounts for the reduced elasticity and breaking strength of scar tissue compared with the intact connective tissue [52].”
• Wound healing: cellular mechanisms and pathological outcomes (2020, background): “Proteoglycans aid construction of mature, cross-linked collagen fibrils and act as a conduit for cell migration [86].”
• Extracellular Matrix Reorganization During Wound Healing and Its Impact on Abnormal Scarring (2015, neutral): “10 ECM proteins like fibronectin, collagens, proteoglycans, heparin and heparin sulphate, bind many growth factors, such as fibroblast growth factors, TGF-b , vascular endothelial growth factor, epidermal growth factor, and bone morphogenetic proteins.”