Thioredoxins, Glutaredoxins, and Peroxiredoxins—Molecular Mechanisms and Health Significance: from Cofactors to Antioxidants to Redox Signaling

By E. Hanschmann, J. R. Godoy, C. Berndt, C. Hudemann, C. Lillig. Antioxidants and Redox Signaling, 2013

640 citations recorded by the source corpus.

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

Botulinum Neurotoxins: Biology, Pharmacology, and Toxicology (2017, background): “There is also an increasing use of BoNT for the treatment of a variety of animal pathologies, including the rather common horse laminitis and dog prostate disorders (Chuang et al., 2006; Lin et al., 2007; Carter and Ben Renfroe, 2009; Hardeman et al., 2013).”
Mechanisms of activation of the transcription factor Nrf2 by redox stressors, nutrient cues, and energy status and the pathways through which it attenuates degenerative disease (2015, positive): “In addition to the GSH and TXN systems, cells utilize the per-oxiredoxin (PRDX) family as cellular antioxidants [31, 32].”
Role of Glutathione in Cancer: From Mechanisms to Therapies (2020, background): “The reversible glutathionylation/deglutathionylation reactions catalyzed by the glutaredoxin (GRX) enzymes also directly use GSH [13,14].”