Interplay of cadherin-mediated cell adhesion and canonical Wnt signaling.

By Julian Heuberger, W. Birchmeier. Cold Spring Harbor Perspectives in Biology, 2010

647 citations recorded by the source corpus.

Subfields: Molecular Biology

View Paper: https://www.semanticscholar.org/paper/70b5dc5d04682f0c46ae37c69568abe775144d7b

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In Molecular Biology

1. T. Valenta
2. G. Hausmann
3. Konrad Basler
4. Gautam Sethi
5. Hans Clevers

Citations in the corpus

• The many faces and functions of β‐catenin (2012, positive): “Reduction of E-cadherin is often connected with increased free cytoplasmic levels of b -catenin, which could translate into increased b - catenin transcription if it escapes cytoplasmic degradation (Heuberger and Birchmeier, 2010).”
• The E-Cadherin and N-Cadherin Switch in Epithelial-to-Mesenchymal Transition: Signaling, Therapeutic Implications, and Challenges (2019, background): “Another mechanism for cadherins to regulate Wnt signaling is via utilizing proteases such as caspase 3, presenilin, ADAM10 or calpain to cleave E- and N-cadherin so that β-catenin will be released to activate Wnt/β-catenin target genes [143].”