In vivo effects of bisphenol A in laboratory rodent studies.
By C. Richter, L. Birnbaum, F. Farabollini, R. Newbold, B. Rubin, C. Talsness, J. Vandenbergh, D. Walser-Kuntz, F. V. vom Saal. Reproductive Toxicology, 2007
1,187 citations recorded by the source corpus.
Subfields: Genetics, Health, Toxicology and Mutagenesis
View Paper: https://www.semanticscholar.org/paper/9508d3d28c92da05c77ad0bc60145e85e5037a36
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Citations in the corpus
- Bisphenol S and F: A Systematic Review and Comparison of the Hormonal Activity of Bisphenol A Substitutes (2015, background): “In the case of BPS and BPF, these chemicals are structural analogs to BPA (Figure 1); thus their effects in physiological systems may be similar.”
- Chapel Hill bisphenol A expert panel consensus statement: integration of mechanisms, effects in animals and potential to impact human health at current levels of exposure. (2007, background): “[3] Richter CR, Birnbaum LS, Farabollini F, Newbold RR, Rubin BS, Talsness”
- Metabolism Disrupting Chemicals and Metabolic Disorders (2016, positive): “sex differences in behavior, thus interfering with normal sexual differentiation of the brain [444-447] which can also affect metabolic processes.”
- Bisphenol A exposure, effects, and policy: a wildlife perspective. (2012, background): “, 2008), defects in male and female reproductive organs (Richter et al., 2007), prostate effects, and increases in malignancies (Hunt et al.”
- Components of plastic: experimental studies in animals and relevance for human health (2009, background): “Also, all hormones show responses that are nonmonotonic , and non-monotonic dose–response curves have been reported in many BPA studies (Richter et al. 2007a,b; Alonso-Magdalena et al. 2008).”
- Bisphenol A and human health: a review of the literature. (2013, background): “Further, BPA can act as an antiestrogen, 19 blocking the estrogenic response by competing with endogenous E2 [5, 6].”
- Endocrine-disrupting chemicals: an Endocrine Society scientific statement. (2009, neutral): “placenta (143, 144) and bind -fetoprotein (the estrogen-”
- Exposure of the U.S. Population to Bisphenol A and 4-tertiary-Octylphenol: 2003–2004 (2007, background): “…vaginal morphology and estrous cycles, disruption of sexual differentiation in the brain, and accelerated growth and puberty (Durando et al. 2007; Howdeshell et al. 1999; Kubo et al. 2003; Richter et al. 2007; Rubin et al. 2006; Schonfelder et al. 2002; Timms et al. 2005; vom Saal et al. 1998).”
- Hormones and endocrine-disrupting chemicals: low-dose effects and nonmonotonic dose responses. (2012, neutral): “been given some attention by bench scientists (29), more”
- Urinary, Circulating, and Tissue Biomonitoring Studies Indicate Widespread Exposure to Bisphenol A (2010, positive): “…[Online 24 March 2010] 1056 volume 118 | number 8 | August 2010 • Environmental Health Perspectives altered body weight, altered organization of the mammary gland, and cancers of the mammary gland and prostate; > 40 of these studies examined doses less than the RfD (Richter et al. 2007).”