TNF and ROS Crosstalk in Inflammation.
By Heiko Blaser, Catherine Dostert, T. Mak, D. Brenner. Trends in Cell Biology, 2016
910 citations recorded by the source corpus.
Subfields: Immunology, Molecular Biology
View Paper: https://www.semanticscholar.org/paper/9417e76688a45d8d5ae163dfb959dd6b55bab4f8
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- Bee Ling Tan
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- Heshu Sulaiman Rahman
- Byong-hee Lee
Citations in the corpus
- Bioactive Peptides (2017, background): “Since reactive oxygen species can cause inflammation [175], lunasin could be important in treating or preventing inflammatory diseases.”
- Designer exosomes produced by implanted cells intracerebrally deliver therapeutic cargo for Parkinson’s disease treatment (2018, neutral): “…brain of mice we con fi rmed the attenuation of ROS-triggered neuroin fl ammation by designer exosomes containing the catalase cargo by pro fi ling the expression levels of several markers (GFAP 28, 43 – 45 , Iba1 45, 46 , TNF α 44, 47 , CD11b 28 ) associated with neuroin fl ammation in the brain (Fig.”
- Antioxidant and Oxidative Stress: A Mutual Interplay in Age-Related Diseases (2018, background): “While, some of the reactive species produced from PGs metabolism may exacerbate inflammation and induce tissue damage (Blaser et al., 2016).”