Differential requirements for Runx proteins in CD4 repression and epigenetic silencing during T lymphocyte development.

By I. Taniuchi, Motomi Osato, Takeshi Egawa, M. Sunshine, S. Bae, T. Komori, Yoshiaki Ito, Dan R. Littman. Cell, 2002

782 citations recorded by the source corpus.

Subfields: Immunology, Molecular Biology

View Paper: https://www.semanticscholar.org/paper/91f3743360a611abae8e9131fd02ea48eb4075a1

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In Immunology

1. Shimon Sakaguchi
2. Tomoyuki Yamaguchi
3. T. Nomura
4. M. Ono
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6. Alexander Y. Rudensky
7. E. Shevach

Citations in the corpus

• The transcription factor PLZF (Zbtb16) directs the effector program of the NKT cell lineage (2008, background): “…et al., 2005), Gata-3 (Kim et al., 2006), and T-bet (Matsuda et al., 2006) showed partial defects of NKT cells as well as T cells and NK cells (Egawa et al., 2007; Intlekofer et al., 2005; Lacorazza et al., 2002; Pai et al., 2003; Samson et al., 2003; Taniuchi et al., 2002; Townsend et al., 2004).”
• T-bet regulates the terminal maturation and homeostasis of NK and Valpha14i NKT cells. (2004, neutral): “1 clone PK136, CyChrome-labeled anti-TCR clone H57-(Hayashi et al., 2001; Taniuchi et al., 2002).”
• Regulatory T cells and immune tolerance. (2008, background): “AML1/Runx1 is crucial for normal hematopoiesis including thymic T cell development (Taniuchi et al., 2002).”