The control of DNA repair by the cell cycle
By Nicole Hustedt, D. Durocher. Nature Cell Biology, 2016
664 citations recorded by the source corpus.
Subfields: Cell Biology, Molecular Biology
View Paper: https://www.semanticscholar.org/paper/918e6c60b8471b14f371ee944a79ffc9f7c64a7c
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Citations in the corpus
- Mitotic CDK promotes replisome disassembly, fork breakage, and complex DNA rearrangements (2018, background): “Our observation that broken forks appear 461 to be processed primarily by SSA and TMEJ is consistent with the finding that HR and 462 NHEJ are inhibited in mitosis (Figure S3A and (Hustedt and Durocher, 2016; Ochs et 463 al., 2016; Peterson et al., 2011)).”
- ATM, ATR, and DNA-PK: The Trinity at the Heart of the DNA Damage Response. (2017, background): “A second, RIF1, interacts with 53BP1 independently of PTIP but in a manner requiring phosphorylation of the 53BP1 N terminus by ATM; ensuing 53BP1 complexes containing PTIP, RIF1, REV7/MAD2L2, and other factors promote DSB repair by NHEJ through mechanisms that are not yet clear (Hustedt and Duroche”