Anxiety and affective style: role of prefrontal cortex and amygdala.

By Richard J. Davidson. Biological Psychiatry, 2002

1,129 citations recorded by the source corpus.

Subfields: Cognitive Neuroscience, Experimental and Cognitive Psychology

View Paper: https://www.semanticscholar.org/paper/9110aa13ada201344920af9f82ebac195dfae31b

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In Cognitive Neuroscience

In Experimental and Cognitive Psychology

Citations in the corpus

Functional neuroanatomy of aversion and its anticipation (2006, methodology): “To assess relations between the right DLPFC and withdrawalrelated unpleasant affect ( Davidson, 2002; Nitschke and Heller, 2002) and between the OFC and both pleasant and unpleasant affect (Rolls, 1999; Nitschke et al., 2004), areas activated by aversive stimuli were correlated with the Negative Af”
The Role of Sleep in Cognition and Emotion (2009, background): “Intriguingly, a similar pattern of anatomical dysfunction has been implicated in a number of psychiatric mood disorders that express cooccurring sleep abnormalities (Davidson 2002; Davidson et al. 2002; New et al. 2007), directly raising the issues of whether such factors (sleep loss and clinical mo”
Prefrontal-subcortical pathways mediating successful emotion regulation. (2008, positive): “These models have established the prefrontal cortex (PFC) as a key player in the cognitive regulation of emotion (Davidson, 2002; Ochsner and Gross, 2005).”
Failure to Regulate: Counterproductive Recruitment of Top-Down Prefrontal-Subcortical Circuitry in Major Depression (2007, positive): “Although abnormalities in the neural circuitry supporting adap-tiveregulationofemotionmayplayadecisiveroleindeterminingvulnerabilitytomajordepressivedisorder(MDD)(Davidson, 2002),littleresearchhasbeenconductedtoexplicitlyexamine thispossibility.”
Neural Bases of Motivated Reasoning: An fMRI Study of Emotional Constraints on Partisan Political Judgment in the 2004 U.S. Presidential Election (2006, background): “As can be seen in Figure 3, processing emotionally threatening information about one’s preferred candidate relative to a neutral target activated distributed sites in the medial PFC, including a large cluster of activation that included the ventral (‘‘affective’’) subdivision of the ACC (centered at”
Neural correlates of behavioral preference for culturally familiar drinks. (2004, background): “Lesions to the DLPFC are Experimental Procedures also known to result in depression (Davidson, 2002), which is hypothesized to result from a decreased ability A total of 67 subjects participated in the study (38 male, 29 female; to use positive affect to modify behavior (Mineka et al., aged 19–50…”
Enhanced visual motion perception in Major Depressive Disorder (2009, background): “…may contribute to the patients’ difficulty with the low-contrast small stimuli— while similar difficulties at a cognitive level may underlie certain cognitive biases associated with MDD, such as an inability to inhibit negative or aversive memories (Davidson, 2002; Goeleven et al., 2006).”
The Set Point Theory of Well-Being Has Serious Flaws: On the Eve of a Scientific Revolution? (2010, methodology): “Drawing on the classic mood research of Wessman and Ricks (1966); (see also Davidson 1992, 2002 ), she proposes that some people have higher emotional reactivity than others and that, if such people are exposed to either particularly favourable or particularly adverse life events, their set-points ”
Chronic Pain and the Emotional Brain: Specific Brain Activity Associated with Spontaneous Fluctuations of Intensity of Chronic Back Pain (2006, background): “It is involved in persistence of emotion after the offset of an elicitor (elicitor in our case is the nociceptive input), and modulates the time course of emotional response, particularly recovery time (Davidson, 2002).”
The Role of Sleep in Emotional Brain Function (2014, background): “…the mechanisms underlying affective brain processes (Critchley 2005, Delgado et al 2006, Hartley & Phelps 2010, Ochsner et al 2009), translationally bridging animal models of emotion regulation and relevant clinical disorders (Davidson 2002, Delgado et al 2006, Drevets et al 2008, Etkin 2010).”
The Amygdala and Persistent Pain (2004, background): “…in emotionality, the emotional evaluation of sensory stimuli, emotional learning, and memory, as well as affective disorders, including anxiety and depression (Davis 1998; Gallagher and Schoenbaum 1999; Maren 1999; Aggleton 2000; LeDoux 2000; Cardinal and others 2002; Davidson 2002; Zald 2003).”
Anxiety-related Bias in the Classification of Emotionally Ambiguous Facial Expressions (2002, background): “Davidson (2002) reviewed the evidence for the involvement of the prefrontal cortex and amygdala in emotion and proposed that there are individual differences in anxiety-related affective styles that are on a continuum.”
Personality Neuroscience and the Biology of Traits (2010, background): “The brain’s right hemisphere appears to be preferentially involved in emotions and motivational states associated with withdrawal, whereas the left hemisphere is preferentially involved in approach (Davidson, 2002).”
Visual Attention and Brain Processes That Underlie Expert Performance: Implications for Sport and Military Psychology (2008, background): “It is well established that the prefrontal cortex has extensive anatomical connections with subcortical limbic structures, particularly the amygdalae, although the communication is indirectly achieved via the anterior cingulate region ( Davidson, 2002, 2004 ).”
Triadic model of the neurobiology of motivated behavior in adolescence (2005, background): “Anxiety Traditional (Gray, 1970) and contemporary (Davidson, 2002; Corr, 2004; McNaughton & Corr, 2004) conceptualizations of normal and pathological anxiety emphasize processes and neural systems involved in motivated behavior.”
What does the prefrontal cortex "do" in affect: perspectives on frontal EEG asymmetry research. (2004, background): “Some researchers have suggested, based upon this anatomical arrangement, that one function of at least certain regions of PFC is to modulate or inhibit activity in the amygdala. Davidson (2000, 2002) has”