Triadic model of the neurobiology of motivated behavior in adolescence

By Monique Ernst, Daniel S. Pine, Michael G. Hardin. Psychological Medicine, 2005

717 citations recorded by the source corpus.

Subfields: Cellular and Molecular Neuroscience, Clinical Psychology, Cognitive Neuroscience

View Paper: https://www.semanticscholar.org/paper/f390750347e591e9152ad708c8074f5edf0e75e8

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In Cellular and Molecular Neuroscience

In Clinical Psychology

In Cognitive Neuroscience

Citations in the corpus

Being Left Out: Neurobiological Correlates of Social Rejection and their Developmental Trajectory (2011, positive): “According to the Triadic model of Ernst et al. (2006), increased risk-taking behavior during adolescence is related”
Gastric distention activates satiety circuitry in the human brain (2008, positive): “The amygdala plays a central role in the processing and memory of emotional reactions, which is important in the establishment of conditioned responses and in the response to novel stimuli (Ernst et al., 2006; Quirk and Gehlert, 2003).”
Juvenile Justice Policy and Practice: A Developmental Perspective (2015, background): “…pronounced during the phase of reward receipt [and thus] favors the hypothesis that overactive reward-related circuitry and immature PFC circuitry potentially bias adolescents toward taking risks [see also Ernst, Pine, and Hardin 2006; Galvan et al. 2006; Casey, Getz, and Galvan 2008]” (p. 66).”
Biological substrates of emotional reactivity and regulation in adolescence during an emotional go-nogo task. (2008, background): “This imbalance may play a role in the increased risk for affective disorders during this period (7,8,9,10).”
Adolescent risky decision-making: Neurocognitive development of reward and control regions (2010, background): “…models of adolescent risk-taking, which suggest an imbalance between the maturation of control and reward brain circuitry (Casey et al., 2008; Ernst et al., 2006; Steinberg et al., 2008), we predicted that participants would choose the high-risk gamble more often when the reward associated…”
Impulsivity as a vulnerability marker for substance-use disorders: review of findings from high-risk research, problem gamblers and genetic association studies. (2008, background): “…studies, which have demonstrated increased ventral striatum, diminished amygdala, and less-efficient frontal cortex activation in adolescents compared to young adults, during the performance of tasks assessing response inhibition and cognitive impulsivity (Ernst et al., 2006a; Eshel et al., 2007).”
The Adolescent Brain (2008, background): “Adolescence is the period between childhood and adulthood encompassed by changes in physical, psychological, and social development (Ernst et al. 2006).”