Systematic meta-analyses and field synopsis of genetic association studies in schizophrenia: the SzGene database

By Nicole C. Allen, Sachin Bagade, M. McQueen, John P. A. Ioannidis, F. Kavvoura, M. Khoury, Rudolph E. Tanzi, Lars Bertram. Nature Genetics, 2008

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The dopamine hypothesis of schizophrenia: version III--the final common pathway. (2009, neutral): “Rather, in common with many other complex diseases, there are a number of genes each of small effect size associated with schizophrenia.(63) The”
From Revolution to Evolution: The Glutamate Hypothesis of Schizophrenia and its Implication for Treatment (2012, background): “In addition, positive genetic associations are reported between both NR1 (Begni et al, 2003) and NR2B polymorphisms and schizophrenia (Martucci et al, 2006; Qin et al, 2005; Allen et al, 2008), suggesting that abnormalities of NMDAR expression or function may directly contribute to schizophrenia.”
Multiplex gene editing by CRISPR-Cpf1 through autonomous processing of a single crRNA array (2016, background): “Mutations in these genes have been implicated in mental illness including schizophrenia and autism spectrum disorders (Allen et al., 2008; Amir et al., 1999; Jamain et al., 2003).”
Animal models of schizophrenia (2011, background): “…protein 1 (DTNBP1) currently thought to be one of the most promising candidate genes for schizophrenia susceptibility, mutations of which show strong correlations with schizophrenia susceptibility and onset in patients (Williams et al., 2005; Allen et al., 2008a; van den Buuse et al., 2009).”