The dopamine hypothesis of schizophrenia: version III--the final common pathway.
By Oliver D. Howes, Shitij Kapur. Schizophrenia bulletin, 2009
2,685 citations recorded by the source corpus.
Subfields: Cellular and Molecular Neuroscience, Psychiatry and Mental health
View Paper: https://www.semanticscholar.org/paper/67326419c18c09fd2d92181a0db983691cc63d68
How researchers see this paper
The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.
In Cellular and Molecular Neuroscience
- Karl J. Friston
- Kent C. Berridge
- Terry E. Robinson
- Klaas E. Stephan
- R. McCutcheon
- J. Stone
- ChrisD . Frith
- Torben Ott
In Psychiatry and Mental health
- Karl J. Friston
- Kent C. Berridge
- R. McCutcheon
- Klaas E. Stephan
- Terry E. Robinson
- Rick A Adams
- J. Stone
- ChrisD . Frith
Citations in the corpus
- Liking, Wanting and the Incentive-Sensitization Theory of Addiction (2016, background): “This dopamine-related fearful salience has been suggested to contribute to human paranoia symptoms in schizophrenia (Barch, Treadway, & Schoen, 2014; Heinz & Schlagenhauf, 2010; Howes & Kapur, 2009) and in psychostimulant-induced psychosis (Cicero, Docherty, Becker, Martin, & Kerns, 2014).”
- A genetically-encoded fluorescent sensor enables rapid and specific detection of dopamine in flies, fish, and mice (2018, neutral): “, 1995), schizophrenia (Howes and Kapur, 2009), and Parkinson’s disease (Lotharius and Brundin, 2002).”
- Glutamate and dopamine in schizophrenia: an update for the 21st century (2015, background): “The combination of this aberrant salience model with the dopaminergic abnormalities described above, and evidence on the impact of environmental and neurodevelopmental risk factors has contributed to the dopamine hypothesis being revised (Howes and Kapur, 2009; Howes and Murray, 2014).”
- The Computational Anatomy of Psychosis (2013, background): “• Second, could the elevated presynaptic striatal dopamine in the psychotic state (Howes and Kapur, 2009) reflect (or compensate for) a primary decrease in prefrontal precision due to NMDAR hypofunction?”
- Dysconnectivity in schizophrenia: where are we now? (2011, background): “To date, there remain substantial divisions within the SZ esearchfieldas to theaetiologyof the illness and,more specifically, recisely how it originates and develops (Fatemi and Folsom, 2009; owes and Kapur, 2009; Stephan et al., 2009a).”
- Dopamine and Cognitive Control in Prefrontal Cortex. (2019, background): “The attractor hypothesis of schizophrenia parallels a suggestion that the positive symptoms in schizophrenic patients are caused by dopamine assigning aberrant salience to sensory stimuli [188].”