Cardioprotective function of inducible nitric oxide synthase and role of nitric oxide in myocardial ischemia and preconditioning: an overview of a decade of research.

By R. Bolli. Journal of Molecular and Cellular Cardiology, 2001

622 citations recorded by the source corpus.

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

Mechanisms underlying acute protection from cardiac ischemia-reperfusion injury. (2008, background): “has been shown to have an important role in PC and cardioprotection (28, 121).”
Interaction of Cardiovascular Risk Factors with Myocardial Ischemia/Reperfusion Injury, Preconditioning, and Postconditioning (2007, background): “…delayed preconditioning has been shown to protect against many consequences of ischemia/reperfusion, including attenuation of early morphological changes, limiting of infarct size, attenuation of stunning and suppression of arrhythmias (Baxter and Yellon, 1998; Bolli, 2001; Baxter, 2002).”
Cytoprotective effects of nitrite during in vivo ischemia-reperfusion of the heart and liver. (2005, neutral): “Additionally, studies investigating NO and NO-releasing agents under in vitro conditions of I/R have consistently reported deleterious effects of NO (32), in contrast to in vivo studies of I/R that have reported beneficial effects of NO therapy (22, 23).”