Neonatal leptin treatment reverses developmental programming.

By M. Vickers, Peter D. Gluckman, A. H. Coveny, P. Hofman, W. Cutfield, A. Gertler, B. Breier, M. Harris. Endocrinology, 2005

618 citations recorded by the source corpus.

Subfields: Endocrine and Autonomic Systems, Pediatrics, Perinatology and Child Health, Physiology

View Paper: https://www.semanticscholar.org/paper/891b199b787cf47fb970bcd1328aed22dca345aa

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Pediatrics, Perinatology and Child Health

1. Mark A. Hanson
2. Peter D. Gluckman
3. M. Holmes
4. J. Seckl
5. Alan Beedle
6. C. Kuzawa
7. E. Sweet
8. Barry M. Popkin

In Physiology

1. Mark A. Hanson
2. Peter D. Gluckman

Citations in the corpus

• Ten Putative Contributors to the Obesity Epidemic (2009, neutral): “This last observation, together with our observation of the protective effects of neonatal leptin treatment (Vickers et al., 2005), confirms the importance of the neonatal period in the programming of obesity, at least in rodents.”
• Epigenetics and the embodiment of race: Developmental origins of US racial disparities in cardiovascular health (2009, background): “…hormone that signals energy status) reverses the adverse metabolic changes triggered by prenatal protein restriction (Gluckman et al., 2007b; Vickers et al., 2005), while orally administered leptin in suckling rats (perhaps mimicking lactation in a well-nourished mother) protects the…”
• Early life events and their consequences for later disease: A life history and evolutionary perspective (2007, background): “1; Vickers et al., 2005) argues for a single mechanism underpinning a broad range of developmental effects.”
• Child health, developmental plasticity, and epigenetic programming. (2011, background): “Indeed, the initial data that gave support to the DOHaD phenomenon were gathered by experimental replication (412, 413).”
• Mechanisms of Disease: glucocorticoids, their placental metabolism and fetal 'programming' of adult pathophysiology (2007, background): “Leptin administration to the mother reverses the effects of dietary restriction on placental 11β-HSD2 and also prevents some of the metabolic sequelae of prenatal malnutrition”