Endothelial Cell Senescence in Human Atherosclerosis: Role of Telomere in Endothelial Dysfunction

By T. Minamino, H. Miyauchi, Toshihiko Yoshida, Y. Ishida, H. Yoshida, I. Komuro. Circulation, 2002

1,069 citations recorded by the source corpus.

Subfields: Cultural Studies, Political Science and International Relations, Sociology and Political Science

View Paper: https://www.semanticscholar.org/paper/8842f9629fd00d3035a561440556227a4fc04a2a

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1. Rafik Amir Salama
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Citations in the corpus

• The essence of senescence. (2010, background): “For example, atherosclerosis is accompanied by an induction of SA-b-GAL activity (Minamino et al. 2002) and telomere shortening (Foreman and Tang 2003).”
• Endothelial Dysfunction in Atherosclerotic Cardiovascular Diseases and Beyond: From Mechanism to Pharmacotherapies (2021, neutral): “2) (Minamino et al., 2002; van der Feen et al., 2020).”
• Cellular Senescence: Aging, Cancer, and Injury. (2019, background): “VSMCs and endothelial cells from human atherosclerotic plaques upregulate a number of senescence markers including SA- -gal, p16, and p21 (133, 223, 229, 336).”
• DNA damage response and cellular senescence in tissues of aging mice (2009, background): “Examples are vascular aging and atherosclerosis (Fenton et al., 2001; Vasile et al., 2001; Minamino et al., 2002), liver cirrhosis (Wiemann et al.”
• Cellular senescence and its effector programs (2014, background): “2008) and agerelated diseased organs in mammals (Minamino et al. 2002; Price et al. 2002).”