Molecular mechanisms involved in the regulation of the endothelial nitric oxide synthase.

By I. Fleming, R. Busse. American Journal of Physiology. Regulatory Integrative and Comparative Physiology, 2003

960 citations recorded by the source corpus.

Subfields: Cell Biology, Molecular Biology, Physiology

View Paper: https://www.semanticscholar.org/paper/8834cd228677638c92733f876367bf0f821e1058

How researchers see this paper

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In Cell Biology

1. U. Förstermann
2. W. Sessa

In Molecular Biology

1. U. Förstermann
2. W. Sessa

In Physiology

1. M. Quon
2. U. Förstermann

Citations in the corpus

• The role of oxidative stress in atherosclerosis. (2009, background): “Under normal conditions, binding to tetrahydrobiopterin (μ∏4), which acts as cofactor and is used as a substrate for L-arginine, leads to NO formation.(18)”
• Cardiovascular actions of insulin. (2007, background): “dephosphorylate eNOS at Ser(1177), whereas PP-1 has been implicated in dephosphorylation of eNOS at Thr(495) (24, 38).”
• Nitric oxide synthases: regulation and function. (2012, background): “This activation is mediated by phosphorylation of the enzyme.(67,68) The eNOS protein can be phosphorylated on several serine (Ser), threonine (Thr), and tyrosine (Tyr) residues.”