Temporal and spatial dynamics of cerebral immune cell accumulation in stroke.
By M. Gelderblom, F. Leypoldt, K. Steinbach, D. Behrens, Chi-un Choe, Dominic A. Siler, T. Arumugam, Ellen Orthey, C. Gerloff, E. Tolosa and 1 more. Stroke, 2009
973 citations recorded by the source corpus.
Subfields: Epidemiology, Neurology
View Paper: https://www.semanticscholar.org/paper/8001b16291e10fd22ee49bc44a7c478452fa36f7
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Citations in the corpus
- Monocyte and macrophage differentiation: circulation inflammatory monocyte as biomarker for inflammatory diseases (2014, background): “It occurs 4 hours after stroke and reaches maximum infiltration in 7 days [56].”
- The biphasic function of microglia in ischemic stroke (2017, neutral): “Cells identified as CD45low CD11b+, CD45intermediate CD11b+, or CD45high CD11b+ represents resting microglia, activated microglia, or macrophages, respectively (Gelderblom et al., 2009).”
- Inflammatory Cytokines in Experimental and Human Stroke (2012, background): “This percentage had at 12 hours increased to, respectively, 5% and 20%, and at 24 hours to, respectively, 25% and close to 30%, of the total number of myeloid cells, after pMCAO (Clausen et al, 2008) and tMCAO (Gelderblom et al, 2009).”
- Inflammatory mechanisms in ischemic stroke: role of inflammatory cells (2010, background): “[47] demonstrate that DCs are increased by 20-fold on Day 3 and 12-fold on Day 7 and thus, constituted a substantial proportion of infiltrating cells.”
- Signaling pathways involved in ischemic stroke: molecular mechanisms and therapeutic interventions (2022, background): “During the whole process of inflammation, various immune cells including microglia, macrophages, and T lymphocytes are activated.(216,217) Also, the production of inflammation-related cytokines are stimulated, as well as interferons or chemokines including monocyte chemoattractant protein-1 (MCP-1).”