Empathy examined through the neural mechanisms involved in imagining how I feel versus how you feel pain.

By Philip L. Jackson, Eric Brunet, Andrew N. Meltzoff, Jean Decety. Neuropsychologia, 2006

829 citations recorded by the source corpus.

Subfields: Cognitive Neuroscience, Psychiatry and Mental health, Social Psychology

View Paper: https://www.semanticscholar.org/paper/7a85b423fa0031fd1751cbf7efffdb9b5291bc58

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In Cognitive Neuroscience

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Citations in the corpus

A structural neural deficit in adolescents with conduct disorder and its association with lack of empathy (2007, background): “There is increasing evidence for a crucial involvement of anterior insula in the experience of empathy (Carr et al., 2003; Jackson et al., 2005, 2006; Singer et al., 2004, 2006; Wicker et al., 2003), but little is known about the role of this structure in aggressive behaviour.”
Attention and reality constraints on the neural processes of empathy for pain (2007, background): “Other recent work observed empathy-related activity in ACC regardless of the modality of stimulation (tactile vs. visual stimulation; Morrison et al., 2004) and body parts where pain stimulation was applied (hand vs. foot; Jackson et al., 2005, 2006a).”
Neuronal correlates of altered empathy and social cognition in borderline personality disorder (2011, positive): “Furthermore, given increased levels of personal distress in patients with BPD (Guttman and Laporte, 2000) where such self-oriented emotional reaction to others is associated with activity in the mid insular cortex (Jackson et al., 2006; Lamm et al., 2007), we expected increased activation in patient”
What Is It like to Feel Another’s Pain?* (2012, background): “Interestingly, recent neuroscientific evidence shows that the process of E-imagining being in pain involves similar activity in the brain as the experience of standard pain (Jackson et al. 2006; Ogino et al. 2007).”
Social functioning in major depressive disorder. (2016, positive): “…activation of the somatosensory-related cortices led to difficulties using social cues for understanding emotional states of others (Keysers et al., 2010), and abnormal activation of the left mACC led to deficits in the ability to evaluate pain processing (Guo et al., 2013; Jackson et al., 2006).”