Mapping auditory hallucinations in schizophrenia using functional magnetic resonance imaging.

By Sukhwinder S. Shergill, M. Brammer, S. Williams, R. Murray, P. McGuire. 2000

645 citations recorded by the source corpus.

Subfields: Cognitive Neuroscience, Psychiatry and Mental health

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Citations in the corpus

Inner Speech: Development, Cognitive Functions, Phenomenology, and Neurobiology (2015, neutral): “…its presence also often activate this brain region, and it is possible that more naturalistic or retrospective forms of symptom capture would reveal more consistent results for alternative regions (van Lutterveld, Diederen, Koops, Begemann, & Sommer, 2013; although see Shergill et al., 2000).”
The rises and falls of disconnection syndromes. (2005, background): “…of combined hodological and topological hyperfunction, with increased activation of Broca’s, Wernicke’s and Geschwind’s territory (Lennox et al., 2000; Shergill et al., 2000) and indirect, diffusion tensor tractography evidence of increased anatomical connectivity between these regions (Hubl et…”
Evidence-based guidelines on the therapeutic use of repetitive transcranial magnetic stimulation (rTMS). (2014, neutral): “During auditory hallucinations, brain areas involved in the perception of speech (primary auditory cortex and associative areas of language in the left hemisphere) show pathological hyperactivity, as determined by neuroimaging studies (Silbersweig et al., 1995; Shergill et al., 2000).”
Oscillatory Cortical Network Involved in Auditory Verbal Hallucinations in Schizophrenia (2012, positive): “Several studies have used this approach [4–14].”
Electrophysiological evidence of corollary discharge dysfunction in schizophrenia during talking and thinking. (2004, background): “However, during the experience of hearing voices when none are spoken (auditory verbal hallucinations), temporal lobe structures are activated, not deactivated in schizophrenics (Dierks et al., 1999, Shergill et al., 2000).”
Genes, dopamine and cortical signal-to-noise ratio in schizophrenia. (2004, background): “Classic symptoms such as auditory hallucinations have been correlated with abnormal activation patterns in sensory association cortices [19] and in prefrontal cortex [ 20 ].”
Decreased language lateralization is characteristic of psychosis, not auditory hallucinations. (2010, background): “…language regions, which is suggestive of a relation between language dys- function and auditory verbal hallucinations (McGuire et al., 1993; Dierks et al., 1999; Shergill et al., 2000, 2001; Van de Ven et al., 2005; Hoffman et al., 2007; Sommer et al., 2007b, 2008a; Diederen et al., 2010).”
Auditory verbal hallucinations predominantly activate the right inferior frontal area. (2008, methodology): “For Permissions, please email: journals.permissions@oxfordjournals.org Downloaded from https://academic.oup.com/brain/article-abstract/131/12/3169/293276 by guest on 26 July 2018 Lennox et al., 1999, 2000; Shergill et al., 2000, 2001; Copolov et al., 2003; Hoffman et al., 2007).”
Exploring the perceptual characteristics of voice-hallucinations in deaf people (2007, background): “Neuroimaging studies of subvocal speech show consistent activation of the areas of the brain used for premotor planning and language processing (the left supplementary motor area, dorsolateral prefrontal cortex and the language association cortices of the temporal lobes) ( Shergill et al., 2000 ).”
Consciousness without a cerebral cortex: A challenge for neuroscience and medicine (2007, result): “…activity elaborating the content of conscious experience in these states are paralleled by corresponding activity changes in the superior colliculus, in contrast to Merker’s testable prediction, although the inferior colliculus was active during auditory hallucinations (Shergill et al. 2000).”