Ruminant adaptation to negative energy balance. Influences on the etiology of ketosis and fatty liver.

By T. Herdt. The Veterinary clinics of North America. Food animal practice, 2000

707 citations recorded by the source corpus.

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

Monitoring and testing dairy herds for metabolic disease. (2004, background): “Cows with type I ketosis typically do not become ketotic before about 3 to 6 weeks in milk [14].”
Invited review: pathology, etiology, prevention, and treatment of fatty liver in dairy cows. (2004, background): “Other risk factors that are associated with decreased feed intake and increased lipolysis in adipose tissue include extended pregnancy and dystocia (Herdt, 2000).”
Feeding- and management-related diseases in the transition cow (2006, background): “A number of reviews have described in detail the metabolic regulation and feed intake around calving (Bauman and Elliot, 1983; Bell, 1995; McNamara, 1995; Bell and Bauman, 1997; Vernon and Pond, 1997; Chilliard, 1999; Herdt, 2000; Ingvartsen and Andersen, 2000; Ingvartsen and Boisclair, 2001), and t”
Invited review: Body condition score and its association with dairy cow productivity, health, and welfare. (2009, background): “When fatty acid mobilization from adipocytes is accelerated, hepatic cells convert excess acetyl CoA generated from β-oxidation into ketone bodies (acetoacetate and BHBA; Herdt, 2000).”
Monitoring metabolic health of dairy cattle in the transition period. (2010, background): “Lower feed intake is associated with increased NEFA which contributes to the risk of fatty liver [21], which in turn is associated with impaired neutrophil function [22].”