Expression of Toll-Like Receptors in Human Atherosclerotic Lesions: A Possible Pathway for Plaque Activation

By K. Edfeldt, J. Swedenborg, G. Hansson, Zhong‐qun Yan. Circulation, 2002

960 citations recorded by the source corpus.

Subfields: Immunology

View Paper: https://www.semanticscholar.org/paper/72a9c54c674fcc5c750ccb5449369300e7def592

How researchers see this paper

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In Immunology

1. G. Hansson
2. A. Hermansson
3. Z. Mallat
4. A. Tedgui
5. Shu Chien
6. J. Chiu
7. Peter Libby
8. R. Medzhitov

Citations in the corpus

• Effects of Disturbed Flow on Vascular Endothelium: Pathophysiological Basis and Clinical Perspectives (2011, background): “responsiveness of ECs to agonists of TLR-2 such as lipopolysaccharide and TNF- , which are augmented in activated ECs of atherosclerotic lesions (166), is blocked by laminar flow, but not by disturbed flow, in a step-flow channel (161).”
• Cytokines in atherosclerosis: pathogenic and regulatory pathways. (2006, background): “TLR1 and TLR2 expression has also been found in human carotid (186) but not in coronary plaques (760).”
• The immune system in atherosclerosis (2011, background): “The endothelium of normal and atherosclerotic arteries expresses a broad repertoire of signaling PRRs, including TLR1, TLR2, TLR3, TLR4, TLR5, TLR7 and TLR9 (refs”