The immune system in atherosclerosis
By G. Hansson, A. Hermansson. Nature Immunology, 2011
2,369 citations recorded by the source corpus.
Subfields: Epidemiology, Immunology
View Paper: https://www.semanticscholar.org/paper/362a2b3f4201e27c16ef4ff1388c7fc972c98dd0
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In Epidemiology
In Immunology
Citations in the corpus
- Stress and cardiovascular disease: an update on current knowledge. (2013, neutral): “Cardiovascular disease risk”
- Oxidized Low-Density Lipoprotein Induces Long-Term Proinflammatory Cytokine Production and Foam Cell Formation via Epigenetic Reprogramming of Monocytes (2014, background): “Subsequent activation of the overlying endothelium leads to the recruitment of circulating monocytes.(1) Monocyte entry”
- NLRP3 Inflammasome and the IL-1 Pathway in Atherosclerosis. (2018, background): “in all stages of atherosclerotic disease development and progression.(14,15)”
- Damage-Associated Molecular Patterns in Inflammatory Diseases (2018, background): “It will be important to define the interacting regions for DAMPs and PRRs for the development of specific inhibitory molecules that can interfere with the interaction and thereby regulate inflammation.”
- Oxidative Stress in Atherosclerosis (2017, background): “Among the SOD enzymes, 3 allele variants of the SOD1 gene (rs9974610, rs10432782, rs1041740) in type 2 diabetics were related to increased death from cardiovascular causes [79]. Polymorphisms in SOD2 resulting in A16V replacement in mitochondrial targeting domain have resulted in an increase in CAD ”
- Western Diet Triggers NLRP3-Dependent Innate Immune Reprogramming (2018, background): “However, it is also well appreciated that disease progression is strongly associated with inflammatory processes involving cells of the innate immune system, mainly monocyte-derived macrophages (Hansson and Hermansson, 2011; Ridker et al., 2017).”