[Mechanisms of hepatic fibrogenesis].

By T. Lamireau, A. Desmoulière, P. Bioulac-Sage, J. Rosenbaum. Archives de pédiatrie, 2002

1,544 citations recorded by the source corpus.

View Paper: https://www.semanticscholar.org/paper/70b4716e2c46a5645d587f65411e2c04f47be2c4

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

1. Jeremy S. Duffield
2. S. Friedman
3. D. Sheppard
4. Fengyun Xu
5. E. Brunt
6. Changwei Liu
7. Dan-dan Zhou
8. Lei Zhang

Citations in the corpus

• Therapy for Fibrotic Diseases: Nearing the Starting Line (2013, background): “Combinations of platelet-derived growth factor (PDGF), connective tissue growth factor (CTGF), and TGFb act on various mesenchymal cell populations to provoke their transdifferentiation into myofibroblasts (37).”
• MicroRNA-122 plays a critical role in liver homeostasis and hepatocarcinogenesis. (2012, unknown): “While low expression of Krüppel-like factor 6 (KLF6) was found primarily in the hepatocytes of WT livers (Figure 7, F and I), stronger KLF6 protein expression was observed in the Mir122a –/– hepatocytes and in some HSCs (Figure 7I, blue arrows).”
• Master Transcription Factors Determine Cell-Type-Specific Responses to TGF-β Signaling (2011, unknown): “TGF-b signaling, through activation of Smad2 and Smad3, plays an essential role in normal development and tissue homeostasis as well as in human diseases from cancer to autoimmunity to cirrhosis (Friedman, 2008; Li and Flavell, 2008; Massagué et al., 2005; Padua and Massagué, 2009; Wandzioch and Zar”
• Hepatocellular carcinoma in non-alcoholic fatty liver disease: an emerging menace. (2012, background): “In addition, activated stellate cells produce various growth factors and may stimulate oncogenic pathways that contribute to the expansion and selection of neoplastic clones of liver cells [93].”
• TGF-β/SMAD Pathway and Its Regulation in Hepatic Fibrosis (2016, background): “In patients with HF, increased concentrations of TGF-β1 correlate with the severity of hepatic fibrosis, suggesting a link between TGF-β expression and progressive liver disease (Friedman 2008; Gressner and Weiskirchen 2006; Lee and Friedman 2011).”