Hydrogen sulfide is an endogenous modulator of leukocyte‐mediated inflammation
By R. Zanardo, V. Brancaleone, E. Distrutti, S. Fiorucci, G. Cirino, J. Wallace. The FASEB Journal, 2006
862 citations recorded by the source corpus.
Subfields: Biochemistry, Immunology, Physiology
View Paper: https://www.semanticscholar.org/paper/6e23f31c141446f828653bef8b20e949708b5d4f
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In Immunology
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Citations in the corpus
- Redox chemistry of biological thiols (2010, background): “Conflicting reports suggest pro- [218] and anti-inflammatory [219], pro- [220] and antiapoptotic [221], and pro- [222] and antioxidant [223] effects of H2S.”
- Hydrogen sulfide and cell signaling. (2011, background): “In the heart, the negative inotropic effect of H2S is antagonized by glibenclamide (20), as is the cardioprotection associated with ischemia/reperfusion injury in isolated hearts (72–74), aspirinmediated leukocyte adherence in mesenteric venules (75), suppression of carrageenan-induced hind-paw edem”
- Prostaglandins, NSAIDs, and gastric mucosal protection: why doesn't the stomach digest itself? (2008, background): “For example, they are both vasodilators and potent inhibitors of leukocyte adherence to the vascular endothelium (45, 226).”
- Hydrogen sulfide is an endogenous stimulator of angiogenesis (2009, background): “Some of the actions of H2S have been attributed to KATP channel opening (7, 10).”
- Physiological implications of hydrogen sulfide: a whiff exploration that blossomed. (2012, methodology): “A recent study showed that injection of rats with H2S donors, NaHS and Na2S, inhibited leukocyte infiltration and adherence to vascular endothelium, and edema formation in a hindpaw edema model induced by the air pouch and carrageenan (755).”
- Protective Actions of H2S in Acute Myocardial Infarction and Heart Failure (2017, neutral): “Lawesson’s Reagent (173) Spontaneous Fast Inflammation”