Hydrogen sulfide is an endogenous stimulator of angiogenesis
By A. Papapetropoulos, A. Pyriochou, Zaid Altaany, Guangdong Yang, A. Marazioti, Zongmin Zhou, M. Jeschke, L. Branski, D. Herndon, Rui Wang and 1 more. Proceedings of the National Academy of Sciences of the United States of America, 2009
827 citations recorded by the source corpus.
Subfields: Biochemistry, Immunology, Physiology
View Paper: https://www.semanticscholar.org/paper/594faafe455c30b7f9e554a12759b78f1d1acd99
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Citations in the corpus
- Physiological implications of hydrogen sulfide: a whiff exploration that blossomed. (2012, background): “Stimulation of endothelial cells with vascular endothelial growth factor (VEGF) increased H2S release, likely due to the upregulation of CSE (467).”
- Protective Actions of H2S in Acute Myocardial Infarction and Heart Failure (2017, neutral): “Multiple signaling pathways are responsible for H2S-induced angiogenesis, including the activation of KATP channels (109,178), PI3K/Akt activation (79), and hypoxia inducible factor-1a (HIF-1a)/VEGF pathways (162).”
- Tumor-derived hydrogen sulfide, produced by cystathionine-β-synthase, stimulates bioenergetics, cell proliferation, and angiogenesis in colon cancer (2013, methodology): “Western blotting for CSE, CBS, and 3-MST was conducted in cell and tissue homogenates as described (3, 5, 7).”
- Hydrogen sulfide and cell signaling. (2011, background): “To date, NaHS has been found to inhibit endothelin-provoked rises in p38 MAPK activity in cultured rat aortic vascular smooth muscle cells (96), yet it upregulates p38 MAPK activity in cultured human umbilical vein endothelial cells (38); this effect was associated with the stimulation of angiogenes”
- Hydrogen sulfide and nitric oxide are mutually dependent in the regulation of angiogenesis and endothelium-dependent vasorelaxation (2012, methodology): “Studies from several laboratories have demonstrated the dependence of the prototypical angiogenic hormone VEGF responses on NO (13, 14) or H2S (15, 16) biosynthesis.”