Research and treatment approaches to depression
By Ma-Li Wong, J. Licinio. Nature Reviews Neuroscience, 2001
690 citations recorded by the source corpus.
Subfields: Behavioral Neuroscience, Clinical Psychology, Social Psychology
View Paper: https://www.semanticscholar.org/paper/6d74c9c6acfc881400353119318395f9d7fbdea9
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In Behavioral Neuroscience
In Clinical Psychology
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Citations in the corpus
- Metalearning and neuromodulation (2002, background): “13 ( Wang & Licinio, 2001 ). The situation depicted in Figure 7(b), where the optimal policy is doing nothing with a small setting of the discount factor γ ,c an be a model of depression.”
- Model organisms: The ascent of mouse: advances in modelling human depression and anxiety (2005, positive): “b–d | Pharmacological antagonism180 (b) or genetic inactivation177 (c,d) of either of the two receptor subunits of the GABAB (γ-aminobutyric acid) receptor (GABAB(1) or GABAB(2)) induces antidepressant-like effects.”
- 5-HTTLPR polymorphism impacts human cingulate-amygdala interactions: a genetic susceptibility mechanism for depression (2005, background): “dtd" [ ]> Depression is among the four leading causes of disability and disease burden throughout the world and is associated with serious medical conditions and mortality across the lifespa”
- In search of a depressed mouse: utility of models for studying depression-related behavior in genetically modified mice (2004, background): “Using genetically modified mice to study depression Depression: still an unmet medical need Depression is one of the most serious disorder in today's society”
- Brain-Derived Neurotrophic Factor Produces Antidepressant Effects in Behavioral Models of Depression (2002, background): “Significant progress has been made in our ability to treat depression, but not all depressed patients respond to available antidepressants, and the therapeutic response requires several weeks or months of treatment (Duman et al., 2000; Wong and Licinio, 2001).”