The cognitive neuroscience of response inhibition: relevance for genetic research in attention-deficit/hyperactivity disorder.

By Adam R. Aron, Russell A. Poldrack. Biological Psychiatry, 2005

623 citations recorded by the source corpus.

Subfields: Agronomy and Crop Science, Food Science

View Paper: https://www.semanticscholar.org/paper/6d1901e1febc720be177f0438d664342d8c44c34

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Agronomy and Crop Science

1. James J. Pekar
2. Daniel J. Simmonds
3. Stewart H. Mostofsky
4. Adam R. Aron

In Food Science

1. James J. Pekar
2. Daniel J. Simmonds
3. Stewart H. Mostofsky
4. Adam R. Aron

Citations in the corpus

• Response inhibition in the stop-signal paradigm (2008, background): “By contrast, in adults with ADHD, SSRT is impaired but go RT is not, indicating a selectivedeficitininhibition.TheinhibitorydeficitADHDis linked to functional and structural differences in the frontobasal-ganglia inhibitory network (for reviews, see Refs [ 42 ,43]).”
• Probing Compulsive and Impulsive Behaviors, from Animal Models to Endophenotypes: A Narrative Review (2010, background): “These include a tendency to pre-potent motor disinhibition, measured by the stop signal reaction time (SSRT) task (Aron and Poldrack, 2005), mediated in human beings through activation of right inferior frontal (RIF) cortex and its subcortical connections (Rubia et al, 2003) and modulated in rats…”
• Meta-analysis of Go/No-go tasks demonstrating that fMRI activation associated with response inhibition is task-dependent. (2008, methodology): “The commonalitybetweenmotorresponseselectionandresponseinhibitionis furtherreflectedintheexecutivedeficitsassociatedwithADHD, where impairments in both motor response inhibition ( Aron & Poldrack, 2005; Barkley, 1997; Nigg, 1999) and motor response selection/execution (Ben-Pazi, Gross-Tsur, Bergman,”
• Subcortical processes of motor response inhibition during a stop signal task (2008, background): “Because fMRI studies employing a block design were vulnerable to a number of important confounds (Aron and Poldrack, 2005), we focused the discussions on event-related paradigms.”
• Impulsivity as a vulnerability marker for substance-use disorders: review of findings from high-risk research, problem gamblers and genetic association studies. (2008, background): “These deficits are also associated with alterations in dopamine neurotransmission: ADHD is widely treated with psychostimulant drugs that increase dopamine (and noradrenaline) function, and is associated with genetic polymorphisms involved in dopamine regulation, including the DAT1 gene (Aron and Po”
• Body mass correlates inversely with inhibitory control in response to food among adolescent girls: An fMRI study (2010, neutral): “As noted, these different regions are part of a distributed frontal lobe inhibitory control network and have been commonly implicated in response inhibition during go/no-go tasks (Buchsbaum et al., 2005; Liddle et al., 2001; Mostofsky et al., 2003; Simmonds et al., 2008; Aron and Poldrack, 2005; Hor”
• The neural bases of momentary lapses in attention (2006, background): “Because these regions have been implicated in stimulus-triggered reorienting of attentio”
• The neural correlates of attention deficit hyperactivity disorder: an ALE meta-analysis. (2006, background): “Aron and Poldrack (2005) examined the existent literature with a narrower focus on the neural correlates of inhibitory control rather than the broader construct of executive processes.”
• Prefrontal-subcortical pathways mediating successful emotion regulation. (2008, neutral): “First, vlPFC—and especially rvlPFC—is well known as a region critical for cognitive control in general, and response inhibition or the selection of information in particular (Aron and Poldrack, 2005; Badre and Wagner, 2005; Nee et al., 2007; Phillips et al., 2003).”
• Cortical and Subcortical Contributions to Stop Signal Response Inhibition: Role of the Subthalamic Nucleus (2006, background): “Although No-Go activates right IFC just as Stopping does (for review, see Aron and Poldrack, 2005; Buchsbaum et al., 2005), and lesions of monkey homologues of IFC affect No-Go (Iversen and Mishkin, 1970; Petrides, 1986) just as lesions of right IFC affect SSRT in humans (Aron et al., 2003a;…”
• Insights into the neural basis of response inhibition from cognitive and clinical neuroscience. (2009, neutral): “Within psychiatry there is a large research effort aimed at determining whether cognitive markers of brain dysfunction, such as response inhibition, may have utility for indexing genetic risk to psychiatric disorder (Aron and Poldrack, 2005).”