Grouping of brain rhythms in corticothalamic systems.

By Mircea Steriade. Neuroscience, 2006

1,293 citations recorded by the source corpus.

View Paper: https://www.semanticscholar.org/paper/672c40468d7409acf579db860a0147088fdc933b

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In Cognitive Neuroscience

Citations in the corpus

Neural syntax: cell assemblies, synapsembles and readers (2010, neutral): “…short lasting (Engel et al., 2001; Fries, 2005; Gray and Singer, 1989; Whittington et al., 2000; Sirota et al., 2008) and often grouped by slower oscillations (Bragin et al., 1995; Canolty et al., 2006; Chrobak and Buzsáki, 1998; Hasenstaub et al., 2005; Sirota et al., 2008; Steriade, 2006).”
Chronic insomnia as a risk factor for developing anxiety and depression. (2007, background): “9-11 Psychopathological states can be assumed to affect descending corticothalamic activity and thus both the initiation and maintenance of sleep by impairing the thalamic dampening of sensory information that characterizes sleep.”
Reward expectation modulates feedback-related negativity and EEG spectra (2007, methodology): “Lower gamma has been associated with thalamocortical and corticocortical coupling (Steriade, 2006), suggesting enhanced connectivity during wins compared to losses.”
Molecular and electrophysiological evidence for net synaptic potentiation in wake and depression in sleep (2008, background): “dtd" [ ]> The brain is remarkably plastic, and plastic changes occur not only as a result of experience and learning, but even in response to variations in spontaneous activit”
Slow-wave sleep takes the leading role in memory reorganization (2010, background): “1 ) that electrophysiological phenomena linked to SWS, such as sharp wave-ripples, spindles and slow oscillations, coordinately contribute to the transfer of memory information from the hippocampus to the neocortex, a process that is thought to underlie the proposed reorganization of memory represen”
Low-frequency neuronal oscillations as instruments of sensory selection (2008, neutral): “Paradoxically, delta-band oscillations, long considered to index states of deep sleep and/or conditions of brain compromise [10], are at the heart of this phenomenon.”
Sleep spindle detection: crowdsourcing and evaluating performance of experts, non-experts, and automated methods (2014, neutral): “These are significant changes, as spindles result from interactions of several regions of the brain, including the thalamic reticular nucleus, thalamocortical relay neurons, the hippocampus, and the cortex 25, 26 .”
The contribution of sleep to hippocampus-dependent memory consolidation. (2007, background): “Hippocampal-to-neocortical information transfer during SWS is under feed-forward control of the neocortical slow oscillation occurring in humans at a peak frequency of 0.75 Hz. The slow oscillation is generated within neocortical networks [ 65 ] at least partially depending on the previous use of th”
Slow-Wave Sleep-Imposed Replay Modulates Both Strength and Precision of Memory (2014, background): “SWS is characterized by slow oscillations (1–5 Hz) in widespread thalamic and neocortical neurons (Buzsáki, 1996; Steriade, 2006) and coincident sharp-wave ripples in both the hippocampal formation and piriform cortex (Mölle et al., 2006; Manabe et al., 2011).”
Sleep for cognitive enhancement (2014, background): “Specifically, spindles and ripples occur preferentially during the slow oscillation up-state (Steriade, 2006), which is a state of widespread neuronal activity.”