Potential antecedent marker of Alzheimer disease

By Mark A. Mintun, Gina N. LaRossa, Yvette I. Sheline, C. Dence, S. Lee, R. Mach, Chester A. Mathis, Steven DeKosky, John C. Morris. 2006

641 citations recorded by the source corpus.

View Paper: https://www.semanticscholar.org/paper/66f01e9c383dcfc03f00992db43f7b2e30a3c2e6

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Citations in the corpus

The Cortical Signature of Alzheimer's Disease: Regionally Specific Cortical Thinning Relates to Symptom Severity in Very Mild to Mild AD Dementia and is Detectable in Asymptomatic Amyloid-Positive Individuals (2008, methodology): “Individuals were considered PIB negative if their binding potential for 4 cortical regions (prefrontal, lateral temporal, precuneus, and gyrus rectus) was below 0.2 (Fagan et al. 2006; Mintun et al. 2006; Fotenos et al. 2008).”
Tau and Aβ imaging, CSF measures, and cognition in Alzheimer's disease (2016, unknown): “of AD-related pathology in vivo, until recently, have been limited to fibrillar Aβ (4, 5)”
Brain imaging in Alzheimer disease. (2012, background): “4; Kemppainen et al. 2006; Mintun et al. 2006; Edison et al. 2008; Shin et al. 2008; Hedden et al. 2009; Lowe et al. 2009; Maetzler et al. 2009; Wolk et al. 2009; Devanand et al. 2010; Jagust et al. 2010; Rabinovici et al. 2010; Roe et al. 2010; Rowe et al. 2010; Tolboom et al. 2010).”
Amyloid deposition is associated with impaired default network function in older persons without dementia (2009, unknown): “We noted that the PPC region of the default network was particularly vulnerable to early amyloid deposition (Mintun et al., 2006), and speculated that older individuals with evidence of aberrant default network activity might harbor occult amyloid pathology.”
Disruption of large-scale brain systems in advanced aging. (2007, methodology): “Individuals were considered PIBnegative if their binding potential for four critical regions was below 0.2 (Mintun et al., 2006).”
Amyloid Deposition, Hypometabolism, and Longitudinal Cognitive Decline (2012, unknown): “Some studies have reported significant cross-sectional associations, 33,34 whereas others have reported no association 22,23,35 between amyloid-PET measurements and cognitive function.”
The Centiloid Project: Standardizing Quantitative Amyloid Plaque Estimation by PET (2014, methodology): “Magnetic resonance imaging (MRI) was performed at either 1.5T or 3T using previously published methods [7; 14; 15; 20].”
Human apoE isoforms differentially regulate brain amyloid-β peptide clearance (2011, unknown): “On the basis of previous studies (7, 27), individuals with mean cortical binding potential (MCBP) for PIB >0.”
Introduction to Revised Criteria for the Diagnosis of Alzheimer’s Disease: National Institute on Aging and the Alzheimer Association Workgroups (2011, background): “[38] Mintun MA, Larossa GN, Sheline YI, Dence CS, Lee SY, Mach RH,”