Kruppel-like factor 2 regulates thymocyte and T-cell migration

By Corey M. Carlson, B. Endrizzi, Jinghai Wu, Xiaojie Ding, M. Weinreich, E. Walsh, M. Wani, J. Lingrel, K. Hogquist, S. Jameson. Nature, 2006

608 citations recorded by the source corpus.

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

Tissue-Resident Memory T Cells (2014, background): “KLF2 promotes other homing markers involved in recirculation, including CCR7 expression (Carlson et al., 2006), which might promote leukocyte egress fromNLT via CCL19+ andCCL21+ afferent lymphatics.”
Finding a way out: lymphocyte egress from lymphoid organs (2007, background): “T cells deficient in the transcription factor KLF2 show defective thymic egress; KLF2 binds the S1P 1 promoter and is required for efficient expression of S1P 1 mRNA in mature thymocyte”
Krüppel-Like Factor 9 Is Necessary for Late-Phase Neuronal Maturation in the Developing Dentate Gyrus and during Adult Hippocampal Neurogenesis (2009, background): “Krüppel-like factor 9 (Klf-9) is a member of the Krüppel-like factor (Klf) family of evolutionarily conserved zinc finger transcription factors (Kaczynski et al., 2003) that has been implicated in mediating a diverse range of biological processes including stem cell maintenance (Jiang et al., 2008”
THE ALLIANCE OF SPHINGOSINE-1-PHOSPHATE AND ITS RECEPTORS IN IMMUNITY (2008, background): “Transcriptional regulation occurs through the transactivation of the gene encoding S1PR1 by Kruppel-like factor 2 ( KLF2 ), which is a transcription factor containing a zinc-finger domai”