Molecular basis of celiac disease.

By Ludvig M. Sollid. Annual Review of Immunology, 2000

678 citations recorded by the source corpus.

Subfields: Epidemiology, Gastroenterology, Immunology

View Paper: https://www.semanticscholar.org/paper/6149b14e190aa627c889fe9355ef3cce76de8010

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Epidemiology

1. L. Day
2. W. Caspary
3. W. Holtmeier
4. Ludvig M. Sollid
5. D. Raulet

In Gastroenterology

1. L. Day
2. W. Caspary
3. W. Holtmeier
4. Ludvig M. Sollid

In Immunology

1. L. Lanier
2. D. Raulet
3. L. Day
4. W. Caspary
5. W. Holtmeier
6. I. Waldhauer
7. A. Steinle
8. M. Smyth

Citations in the corpus

• Wheat seed storage proteins: Advances in molecular genetics, diversity and breeding applications (2014, background): “Strikingly, most of the gluten peptides implicated in 689 CD require modification by the enzyme transglutaminase before they can bind to the 690 disease-predisposing HLA-DQ molecules and trigger T cell responses ( Koning 2003).”
• Proteins from land plants – Potential resources for human nutrition and food security (2013, background): “Plant proteins have also suffered from an unfavourable image with regard to the allergic reactions they cause in 1e2% of the population and the link with coeliac disease from consuming specific proteins from wheat, barley and rye (Mills, Jenkins, Alcocer, & Shewry, 2004; Sollid, 2000).”
• Coeliac disease: dissecting a complex inflammatory disorder (2002, result): “Although the immunogenetic data are less clear regarding the primary disease-susceptibility determinant of the DR4–DQ8 haplotype, most data favour DQ8 (Ref”
• Coordinated induction by IL15 of a TCR-independent NKG2D signaling pathway converts CTL into lymphokine-activated killer cells in celiac disease. (2004, background): “In addition, NKG2D mediated lysis of gluten-specific DQ2- or DQ8-restricted CD4 T cells is well established in the lamina propria (Koning, 2003; of C1R/MIC but not control cells in TCR CD8 CTL lines cultured with 100 U/ml IL2 or 20 ng/ml IL15 and Sollid, 2000), a key pathologic event is a massive ce”
• Celiac disease (2006, background): “Based on the high association between human leukocyte antigens (HLA) and celiac disease (over 95%) [96], it is thought that HLA-DQ2 positive antigen-presenting cells have gliadin peptides toxic to CD4+ T cells.”