Inflammation
By O. Parnes. The Lancet, 2008
1,466 citations recorded by the source corpus.
Subfields: Molecular Biology
View Paper: https://www.semanticscholar.org/paper/6144acffe711146136da280dc7a43141d6071574
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In Molecular Biology
Citations in the corpus
- Intermuscular Fat: A Review of the Consequences and Causes (2014, background): “Interestingly, several authors have reported relationships between increased proinflammatory cytokines and decreased muscle [83, 84] and mobility function [85–87] that are strikingly similar to those reported between IMAT and muscle and mobility function [19].”
- Autophagy in inflammation, infection, and immunometabolism (2021, background): “In bacterial infections however, the antimicrobial effects are attributable to autophagy whereas type I IFN activation may be an epiphenomenon that is neutral or with immunological consequence by suppressing inflammasome-driven inflammation (Burke et al., 2020; Guarda et al., 2011; Labzin et al., 20”
- NLRP3 inflammasome: From a danger signal sensor to a regulatory node of oxidative stress and inflammatory diseases (2015, background): “The atherogenic process is initiated by recruitment of LDL cholesterol into the arterial wall leading to inflammatory injury and the excess of cholesterol is partially deposited as cholesterol crystals [149,150].”
- Free radicals, metals and antioxidants in oxidative stress-induced cancer. (2006, background): “Activated macrophages initiat increase in oxygen uptake that gives rise to a va of reactive oxygen species, including superoxide an nitric oxide and hydrogen peroxide [10].”
- Hidden Fluid Mechanics: A Navier-Stokes Informed Deep Learning Framework for Assimilating Flow Visualization Data (2018, background): “One of the real-world examples of the above benchmark problem is encountered in cardiovascular fluid mechanics, where one or multiple coronary arteries are partially blocked by atherosclerosis plaques formed by the lipid accumulation [32].”
- Neutrophil: A Cell with Many Roles in Inflammation or Several Cell Types? (2018, background): “NETs can also regulate cytokine production from macrophages in atherosclerosis (Warnatsch et al., 2015), and induce endothelial dysfunction directly by activation and damage of endothelial cells (Knight et al., 2014).”