Adipocytokines: mediators linking adipose tissue, inflammation and immunity

By Herbert Tilg, A. Moschen. Nature reviews. Immunology, 2006

3,236 citations recorded by the source corpus.

Subfields: Endocrine and Autonomic Systems, Epidemiology, Physiology

View Paper: https://www.semanticscholar.org/paper/5f60fed1cbc3412fcf1320a97f9580a42a64ef8a

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Endocrine and Autonomic Systems

1. Sushil Kumar Jain
2. Herbert Tilg

In Epidemiology

1. M. Pinzani
2. F. Bäckhed
3. P. Calder
4. Antonio Gonzalez
5. E. Tsochatzis
6. Ruth E. Ley
7. A. Kaser
8. Rob Knight

In Physiology

1. Rob Knight
2. F. Bäckhed
3. Ruth E. Ley
4. Herbert Tilg
5. Omry Koren
6. S. Salminen
7. E. Isolauri
8. Julia Goodrich

Citations in the corpus

• Dietary factors and low-grade inflammation in relation to overweight and obesity (2011, positive): “(10) Dietary factors that promote inflammation are oxidised lipids, SFA and trans-fatty acids.”
• Gut microbiome, obesity, and metabolic dysfunction. (2011, background): “characterized by low-grade inflammation, and adipokines play a central role (74).”
• Adipokines as emerging mediators of immune response and inflammation (2007, background): “Macrophages are an additional source of soluble mediators and might contribute and perpetuate local and systemic inflammation”
• The multiple-hit pathogenesis of non-alcoholic fatty liver disease (NAFLD). (2016, background): “[83] Tilg H, Moschen AR.”
• Effects of Exercise Training on Cardiorespiratory Fitness and Biomarkers of Cardiometabolic Health: A Systematic Review and Meta-Analysis of Randomized Controlled Trials (2015, background): “Obesity has become widely regarded as a chronic proinflammatory state, and substantial evidence indicates that chronic inflammation in adipose tissues, especially in white adipose tissue, could lead to insulin resistance.(211,212) Consequently, it is biologically plausible that by reducing the white”
• TNF-α and adipocyte biology (2007, neutral): “cies-specific differences in its cellular source [127].”
• Host remodeling of the gut microbiome and metabolic changes during pregnancy. (2012, background): “Metabolic syndrome is a range of phenotypes that increase an individual’s risk of developing type 2 diabetes, including hyper-glycemia, insulin resistance, excess adiposity, and low-grade inflammation (Tilg and Moschen, 2006; Vijay-Kumar et al., 2010).”
• Obesity, Oxidative Stress, Adipose Tissue Dysfunction, and the Associated Health Risks: Causes and Therapeutic Strategies (2015, background): “Both TNF-a and IL-6 increase the activities of NOX and the production of superoxide anion.(62,63) Hyperleptinemia and oxidative stress in obesity.”