Magnetic resonance spectroscopy to measure hepatic triglyceride content: prevalence of hepatic steatosis in the general population.
By L. Szczepaniak, P. Nurenberg, D. Leonard, J. Browning, J. Reingold, S. Grundy, Helen H. Hobbs, R. Dobbins. American Journal of Physiology. Endocrinology and Metabolism, 2005
1,593 citations recorded by the source corpus.
Subfields: Endocrinology, Diabetes and Metabolism, Epidemiology, Hepatology
View Paper: https://www.semanticscholar.org/paper/5ca0ad4a071365250312fc0e9db8e857ff2c65f8
How researchers see this paper
The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.
In Endocrinology, Diabetes and Metabolism
In Epidemiology
- Gerald I. Shulman
- V. Samuel
- Christopher D. Byrne
- G. Targher
- S. Friedman
- R. Loomba
- Y. Saiman
- J. Puche
In Hepatology
- Christopher D. Byrne
- G. Targher
- Gerald I. Shulman
- S. Friedman
- R. Loomba
- Y. Saiman
- J. Puche
- J. Bonner
Citations in the corpus
- Reversal of type 2 diabetes: normalisation of beta cell function in association with decreased pancreas and liver triacylglycerol (2011, background): “It continued to decline throughout the intervention period to reach the normal range for non-obese individuals [20] (2.”
- Quantitative Assessment of Liver Fat with Magnetic Resonance Imaging and Spectroscopy (2011, methodology): “56% fat-fraction as abnormal, based on a subset of 345 patients with no identifiable risk factors for hepatic steatosis(14).”
- NAFLD: a multisystem disease. (2015, background): “S48 Journal of Hepatology 201 regarding the sensitivity and specificity of ultrasonography to detect liver fat, the reader is referred to the following [10–12]; and for information, regarding the sensitivity and reproducibility of magnetic resonance techniques to assess liver fat [13,14].”
- Non-alcoholic fatty liver disease (NAFLD): a review of pathophysiology, clinical management and effects of weight loss (2022, background): “Unfortunately MRS has a higher sensitivity to detect the earlier mentioned pathological processes it is (not yet) widely available [43].”
- Genetic variation in PNPLA3 confers susceptibility to nonalcoholic fatty liver disease (2008, methodology): “Hepatic fat content was measured in the Dallas Heart Study using proton magnetic resonance spectroscopy ( 1 H-MRS), the most accurate, quantitative, noninvasive method availabl”
- Intrahepatic fat, not visceral fat, is linked with metabolic complications of obesity (2009, neutral): “5% of liver volume) (n 10) IHTG content (Table 1) (41).”
- Obesity and nonalcoholic fatty liver disease: From pathophysiology to therapeutics. (2019, neutral): “of obesity and NAFLD, it is not unexpected that the prevalence of NAFLD in obese individuals is higher than that of the general population (25–30% [61,62]), although it varies in different studies according to ethnicity, age, other predisposing factors, including T2DM, and, importantly, the method o”
- Non-invasive assessment and quantification of liver steatosis by ultrasound, computed tomography and magnetic resonance. (2009, methodology): “(1)H MRS is being used in a large population studies to determine the prevalence of non-alcoholic fatty liver disease in the general adult population [97] and also in an increasing number of longitudinal clinical studies [99– 104].”