Human Cytomegalovirus Activates Inflammatory Cytokine Responses via CD14 and Toll-Like Receptor 2

By T. Compton, E. Kurt-Jones, K. Boehme, J. Belko, E. Latz, D. Golenbock, R. Finberg. Journal of Virology, 2003

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Immunobiology of Human Cytomegalovirus: from Bench to Bedside (2009, background): “HCMV has also been demonstrated to activate and signal through the interaction of gB/gH and TLR2, which triggers inflammatory cytokine production (28, 48, 117).”
Pathogen Recognition and Inflammatory Signaling in Innate Immune Defenses (2009, background): “The involvement of TLR-independent recognition systems in the early response to HSV infection has been strongly suggested by several individual reports (127, 223, 301) and further supported by in vivo data demonstrating that mice lacking TLR9 or myeloid differentiation primary-response gene 88 (MyD8”
The Role of TLR2 in Infection and Immunity (2012, neutral): “(2009), Compton et al. (2003) Hepatitis B capsid***** Hepatitis B TLR2/?”
Toll-like receptors in antiviral innate immunity (2013, background): “For the latter, TLR2 has been shown to recognize the glycoproteins B and H of human cytomegalovirus [163,164], the glycoproteins gH/gL and gB of HSV [165], the UTPase of EBV [166], the hemagglutinin protein of measles virus [20], the nsp4 of rotavirus [21], and the core and NS3 proteins of HCV [167]”