The thrifty phenotype hypothesis.
By N. Hales, D. Barker, Hales. British Medical Bulletin, 2001
2,726 citations recorded by the source corpus.
Subfields: Obstetrics and Gynecology, Pediatrics, Perinatology and Child Health
View Paper: https://www.semanticscholar.org/paper/4a7f9ea4dd88c1d1793b79539dc887b93ee88c1c
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In Obstetrics and Gynecology
- Frank B Hu
- Mark A. Hanson
- Peter D. Gluckman
- Linda Partridge
- Alan Beedle
- Lei Chen
- Hamish G. Spencer
- D. Magliano
In Pediatrics, Perinatology and Child Health
- Mark A. Hanson
- Peter D. Gluckman
- P. Monaghan
- Dorret I. Boomsma
- Hamish G. Spencer
- Alan Beedle
- Frank B Hu
- P. Zimmet
Citations in the corpus
- Endocrine regulation of human fetal growth: the role of the mother, placenta, and fetus. (2006, background): “Studies by Barker and Osmond (376, 377) examined the geographical relationship between current death rates from heart disease or stroke and prior infant or maternal mortality rates in England and Wales.”
- Epigenetics and the environment: emerging patterns and implications (2012, background): “A hypothesis that emerged from epidemiological studies proposes that fetal metabolic adjustments to nutritionally unfavourable conditions can affect growth and development, and may substantially alter the risk of chronic disorders later in lif”
- The nutrition transition to 2030. Why developing countries are likely to bear the major burden (2005, background): “@The adverse impacts of the rapid nutrition transition are likely to be compounded by a number of other factors that are specific to developing countries. This may not only mean that the nutrition transition proceeds faster in developing countries, but also that its adverse impacts are likely to be ”
- Paternally-induced transgenerational environmental reprogramming of metabolic gene expression in mammals (2010, background): “…(Horton, 2005), cultural inheritance of stress reactivity and maternal grooming behavior in rats (Meaney et al., 2007; Weaver et al., 2004), and metabolic and psychiatric sequelae of fetal malnutrition in humans and rodents (Hales and Barker, 2001; Harris and Seckl, 2010; Symonds et al., 2009).”
- Classical twin studies and beyond (2002, background): “Studies of the effects of fetal and infant growth on later health (the Barker hypothesis; for example, see Ref”
- Promoting Health and Longevity through Diet: from Model Organisms to Humans (2015, positive): “The thrifty phenotype hypothesis (Hales and Barker, 2001) postulates that many of the changes in organ structure and metabolism seen in humans in response to restricted nutrition—particularly of protein—in utero can be understood as the consequences of immediate”