Molecular Mimicry, Bystander Activation, or Viral Persistence: Infections and Autoimmune Disease
By R. Fujinami, M. V. von Herrath, U. Christen, J. Whitton. Clinical Microbiology Reviews, 2006
658 citations recorded by the source corpus.
Subfields: Cardiology and Cardiovascular Medicine, Public Health, Environmental and Occupational Health, Rheumatology
View Paper: https://www.semanticscholar.org/paper/45ed12b9cb49e82d249d7211396658b4108facd3
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In Public Health, Environmental and Occupational Health
In Rheumatology
Citations in the corpus
- Multiple sclerosis: a complicated picture of autoimmunity (2007, background): “Viral infection can trigger autoimmune T cell reactions either by molecular mimicry or by bystander activatio”
- Environmental risk factors for multiple sclerosis. Part I: The role of infection (2007, background): “The observation that MS tends to occur several years after mononucleosis(57) may also be important, because it suggests that EBV acts as a priming factor or initiator of the pathological process, and that other events may be needed to trigger disease expression.(118) Further clues on how EBV may inc”
- Multiple Sclerosis: Pathogenesis, Symptoms, Diagnoses and Cell-Based Therapy (2016, background): “Environmental factors, including exposure to viral and bacterial agents such as Epstein Barr virus (EBV), human herpes virus type 6, and mycoplasma pneumonia (25), in addition to smoking (26), vitamin deficiency (27), diet (28, 29), and exposure to UV radiation (30) are associated with the onset of ”