The osteoblast: a sophisticated fibroblast under central surveillance.

By P. Ducy, T. Schinke, G. Karsenty. Science, 2000

1,157 citations recorded by the source corpus.

Subfields: Endocrinology, Diabetes and Metabolism, Molecular Biology, Oncology

View Paper: https://www.semanticscholar.org/paper/4398abdccfe835f5e2d02b5e891107e4f56a2c4f

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Endocrinology, Diabetes and Metabolism

1. Stephen Honig
2. P. Rackoff
3. Rui Luís Reis
4. A. Salgado
5. O. Coutinho

In Molecular Biology

1. Stephen Honig
2. P. Rackoff
3. Rui Luís Reis
4. A. Salgado
5. O. Coutinho
6. S. Harada
7. G. Rodan
8. Xu Feng

In Oncology

1. Stephen Honig
2. P. Rackoff
3. Rui Luís Reis
4. A. Salgado
5. O. Coutinho
6. Xu Feng
7. J. McDonald
8. Osbjorn M. Pearson

Citations in the corpus

• Bone tissue engineering: state of the art and future trends. (2004, positive): “The elaboration, maintenance and resorption of this remarkable tissue result from the interaction of three cell types [7-11]: osteoblasts, osteocytes and osteoclasts.”
• Buried alive: How osteoblasts become osteocytes (2006, neutral): “Ducy et al. (2000); 48.”
• Autologous platelets as a source of proteins for healing and tissue regeneration (2003, background): “Bone regeneration requires the recruitment, proliferation and maturation of osteoblasts which are derived from mesenchymal stem cells (MSC) (56, 57).”
• Control of osteoblast function and regulation of bone mass (2003, background): “Osteoblasts are derived from mesenchymal precursor cells that also give rise to chondrocytes, myoblasts, adipocytes and tendon cell”
• Endocrine regulation of energy metabolism by the skeleton. (2007, methodology): “Osteoblasts’ Regulation of b-Cell and Adipocyte Biology That adipocytes regulate bone mass by acting on osteoblasts suggested that osteoblasts may affect adipocyte biology (Ducy et al., 2000a).”
• Disorders of bone remodeling. (2011, background): “MSCs give rise to osteoprogenitors, which differentiate into preosteoblasts and then mature osteoblasts (12).”
• The Female Athlete Triad (2007, positive): “Low energy availability may also suppress bone formation via effects on other hormones, including cortisol and leptin (48,91,113,114,121,128,136,176,206,219).”
• Transforming Growth Factor- 1 to the Bone (2005, background): “Moreover, bone formation is regulated by endocrine factors such as sex steroid hormones, PTH , 1,25dihydroxyvitamin D3 [1,25-(OH)2D3, the active metabolite of vitamin D], and leptin (104, 105).”
• The aging of Wolff's "law": ontogeny and responses to mechanical loading in cortical bone. (2004, background): “Given its complexity, it is not surprising that osteoblast regulation is only very poorly understood (Ducy et al., 2000).”
• Role of receptor activator of nuclear factor-κB ligand and osteoprotegerin in bone cell biology (2001, background): “and/or OPG [65, 89, 90], indicating that there is extensive”