Inhalation vs. aspiration of single-walled carbon nanotubes in C57BL/6 mice: inflammation, fibrosis, oxidative stress, and mutagenesis.
By A. Shvedova, E. Kisin, A. Murray, V. Johnson, O. Gorelik, S. Arepalli, A. Hubbs, R. Mercer, P. Keohavong, N. Sussman and 9 more. American Journal of Physiology - Lung cellular and Molecular Physiology, 2008
640 citations recorded by the source corpus.
View Paper: https://www.semanticscholar.org/paper/40613589e1c21cc69fd4e731f51fa76a63e17eb6
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Citations in the corpus
- Interactions of nanomaterials and biological systems: implications to personalized nanomedicine (2012, background): “Exposure of mice to CNTs by inhalation increased the rate of mutation of the K-ras gene locus in the lung, with the mutations occurring during times of maximum inflammation in the tissue [210].”
- Mechanisms of Nanoparticle-Induced Oxidative Stress and Toxicity (2013, background): “Moreover, NADPH-mediated ROS are critical for SWCNT-induced pulmonary responses [91].”
- In vitro approaches to assess the hazard of nanomaterials (2017, background): “…using in vivo dose equivalents (0.02 to 0.2 μg/cm2), resulted in a dose-dependent transient pulmonary inflammation followed by fibroblast cell proliferation, alveolar wall thickening, and collagen I production culminating in persistent pulmonary fibrosis (Shvedova et al., 2008; Wang et al., 2010).”