Tissue‐specific and inducible Cre‐mediated recombination in the gut epithelium

By F. El Marjou, K. Janssen, Benny Hung‐Junn Chang, Mei Li, V. Hindié, L. Chan, D. Louvard, P. Chambon, D. Metzger, S. Robine. Genesis, 2004

1,068 citations recorded by the source corpus.

Subfields: Genetics, Molecular Biology, Pathology and Forensic Medicine

View Paper: https://www.semanticscholar.org/paper/3c513a2f2098bcac62afac6edf7f1f10f1f787d1

How researchers see this paper

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In Genetics

1. R. Hen
2. Leonard I. Zon
3. T. Ganz

In Molecular Biology

1. R. Hen
2. S. Piccolo
3. Leonard I. Zon
4. G. Karsenty
5. P. Ducy

Citations in the corpus

• The iron exporter ferroportin/Slc40a1 is essential for iron homeostasis. (2005, methodology): “Based on earlier characterization of the vil-Cre-ERT2 mouse model, it is likely that the observed 50% deletion in whole intestine corresponds to 100% deletion in crypt cells and enterocytes, with no deletion in nonepithelial cell types (el Marjou et al., 2004).”
• Lrp5 controls bone formation by inhibiting serotonin synthesis in the duodenum: an entero-bone endocrine axis (2008, background): “…Htr1b / , Htr2a / , Crebfl/fl, Atf4 / , Runx2 / , Osx / , Villin-Cre, and a1(I)Col-Cre mice was previously reported (Dacquin et al., 2002; el Marjou et al., 2004; Glass et al., 2005; Heath and Hen, 1995; Kato et al., 2002; Mantamadiotis et al., 2002; Weisstaub et al., 2006; Yang et…”
• YAP/TAZ-Dependent Reprogramming of Colonic Epithelium Links ECM Remodeling to Tissue Regeneration (2018, background): “…S127A (kindly provided by F. Camargo (Camargo et al., 2007)), Yap1fl/fl (kindly provided by DJ Pan; (Zhang et al., 2010), Villin-CreERT2 (kindly provided by S. Robine; el Marjou et al., 2004), Tazfl/f (Azzolin et al., 2014),RAG2 / (Taconic, US) andRosa26-rtTAM2 (stock #006965, Jackson Laboratory).”
• Mathematical Modelling of Molecular Pathways Enabling Tumour Cell Invasion and Migration (2015, methodology): “In our previous work, this specific double mutation NICD GoF/p53 LoF has been carried out experimentally in a mouse model, by crossing the villin-CreERT2mice [129] (in this study referred as p53 LoF) and RosaN1icmice [130] (in this study referred as NICD GoF) with the isogenic C57BL/6 animals to gen”