The relationship of depression and stressors to immunological assays: a meta-analytic review.
By E. Zorrilla, L. Luborsky, J. McKay, R. Rosenthal, A. Houldin, A. Tax, R. McCorkle, D. Seligman, K. Schmidt. Brain, behavior, and immunity, 2001
1,004 citations recorded by the source corpus.
Subfields: Behavioral Neuroscience, Biological Psychiatry, Public Health, Environmental and Occupational Health
View Paper: https://www.semanticscholar.org/paper/363ef02bc71f4e4d0b62af7ced6362af668e0d2f
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In Behavioral Neuroscience
- Michael R. Irwin
- Andrew H. Miller
- George M. Slavich
- Gregory E. Miller
- Charles L Raison
- L. Capuron
- V. Maletić
- E. Haroon
In Biological Psychiatry
- Andrew H. Miller
- Michael R. Irwin
- George M. Slavich
- Charles L Raison
- Gregory E. Miller
- L. Capuron
- Andrew Steptoe
- V. Maletić
In Public Health, Environmental and Occupational Health
Citations in the corpus
- Immune System to Brain Signaling: Neuropsychopharmacological Implications (2011, background): “…the early findings were not always replicated, meta-analytic approaches to the literature in this area have reached the consensus that statistically reliable decreases in lymphocyte responses to T cell mitogens are apparent in depressed individuals (Zorrilla et al., 2001; Irwin and Miller, 2007).”
- Associations of Depression With C-Reactive Protein, IL-1, and IL-6: A Meta-Analysis (2009, background): “Earlier meta-analyses (23,24) assessed some of these relationships and found positive associations between IL-6 and intensity of depression but did not include outcomes for CRP.”
- Religiousness and depression: evidence for a main effect and the moderating influence of stressful life events. (2003, result): “By comparison, other meta-analyses have found that variables such as self-focused attention (Mor & Winquist, 2002) and immunological parameters (Zorilla et al., 2001) on average correlate with depression in the absolute value range of .20 to .25.”
- Depressive disorders and immunity: 20 years of progress and discovery. (2007, background): “Indeed, depression has been associated with increases in circulating levels of the pro-inflammatory cytokine, IL-6 in adults with major depression (Zorrilla et al., 2001), in depressed elderly populations, and in those depressed persons with chronic medical disorders such as rheumatoid arthritis (Za”
- Psychological stress and the human immune system: a meta-analytic study of 30 years of inquiry. (2004, background): “Zorrilla et al. (2001) reported on 82 studies published between 1980 and 1996, finding potentially adaptive effects of acute stressors in addition to evidence for immunosuppression with longer stressors.”
- From Stress to Inflammation and Major Depressive Disorder: A Social Signal Transduction Theory of Depression (2014, background): “As reviewed elsewhere, elevated proinflammatory cytokine levels are not the only indication that inflammation may be a key biological mediator of depression (Irwin & Miller, 2007; Maes, 2011; Zorrilla et al., 2001).”
- Psychoneuroimmunology Meets Neuropsychopharmacology: Translational Implications of the Impact of Inflammation on Behavior (2012, background): “…revealed that peripheral blood elevations in the cytokines, interleukin (IL)-6, tumor necrosis factor (TNF)-a, and the acute-phase reactant, c-reactive protein (CRP) are some of the most reliable biomarkers of increased inflammation in depressed patients (Zorrilla et al, 2001; Dowlati et al, 2010).”
- A Scientific Review of the Remarkable Benefits of Happiness for Successful and Healthy Living (2013, background): “A review by Zorrilla et al. (2001) found that stress is related to a weaker immune system.”
- Inflammation and Its Discontents: The Role of Cytokines in the Pathophysiology of Major Depression (2009, positive): “When compared with nondepressed individuals, both medically ill and medically healthy patients with major depression have been found to exhibit all of the cardinal features of inflammation, including elevations in relevant inflammatory cytokines and their soluble receptors in peripheral blood and ce”
- Inflammation Causes Mood Changes Through Alterations in Subgenual Cingulate Activity and Mesolimbic Connectivity (2009, positive): “The results of this study illustrate that experimental inflammation induced by typhoid vaccination produces a robust increase in pro-inflammatory cytokines (IL-6) and reduction in a composite measure of mood (that includes both cognitive-mood and neurovegetative contributions) in normal healthy male”