Brain ischemia and reperfusion: molecular mechanisms of neuronal injury.
By B. White, J. Sullivan, D. DeGracia, B. O’Neil, R. Neumar, L. Grossman, J. Rafols, G. Krause. Journal of Neurological Sciences, 2000
821 citations recorded by the source corpus.
Subfields: Cell Biology, Cellular and Molecular Neuroscience, Molecular Biology
View Paper: https://www.semanticscholar.org/paper/361b0e3f196e1408a8c904a3a43acb01a2eb4975
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Citations in the corpus
- Antioxidant Defenses in Fish: Biotic and Abiotic Factors (2005, neutral): “The reperfusion of ischemic organs triggers the over-generation of ROS and induces lipid peroxidation, protein oxidation and DNA damage (Lefer and Grander, 2000; White et al., 2000) due to a lower antioxidant potential of anoxic/ hypoxic mammalian organs (Hermes-Lima and Zenteno-Savı́n, 2002).”
- Diversity of the mammalian sodium/proton exchanger SLC9 gene family (2004, background): “, cardiac arrhythmias, altered synaptic transmission) and ultimately tissue damage, including free radical toxicity, cellular edema, apoptosis and necrosis (reviewed in [61, 111, 159]).”