Epigenetic epidemiology of the developmental origins hypothesis.
By Robert A. Waterland, Karin B. Michels. Annual review of nutrition, 2007
878 citations recorded by the source corpus.
Subfields: Molecular Biology, Pediatrics, Perinatology and Child Health
View Paper: https://www.semanticscholar.org/paper/31663b5ca1a51315d025544a05f8823358993cc4
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In Molecular Biology
In Pediatrics, Perinatology and Child Health
Citations in the corpus
- Epigenetics and the embodiment of race: Developmental origins of US racial disparities in cardiovascular health (2009, background): “…environmental factors can modify epigenetic processes, thereby affecting epigenetic marks and downstream patterns of gene expression in specific cells and cell lineages (Gluckman et al., 2007a,b; Ho and Tang, 2007; Jirtle and Skinner, 2007; Waterland and Jirtle, 2004; Waterland and Michels, 2007).”
- Persistent epigenetic differences associated with prenatal exposure to famine in humans (2008, background): “The developmental origins hypothesis states that adverse conditions during development contribute to adult disease risk (22).”
- Ten Putative Contributors to the Obesity Epidemic (2009, neutral): “Most importantly, transient environmental influences during critical periods of development can induce permanent alterations in epigenetic gene regulation and associated phenotypes (Waterland and Michels, 2007).”
- Child health, developmental plasticity, and epigenetic programming. (2011, background): “Waterland RA, Michels KB 2007 Epigenetic epidemiology of the developmental origins hypothesis.”
- DNA methylation differences after exposure to prenatal famine are common and timing- and sex-specific. (2009, background): “Persistent epigenetic changes induced by environmental factors are a plausible molecular mechanism underlying the relationship between early development and later-life disease (15,16).”
- Epigenetics for behavioral ecologists (2013, background): “1998; Waterland & Michels 2007 Mus musculus (mouse) Aggressive behavior in females flanked by 2 males vs. those flanked by 2 females in utero The effects of intrauterine position are enhanced by maternal stressors such as intense light, heat, and crowding The sex of litter mates and corticosterone…”