Macrophage subsets in atherosclerosis

By G. Chinetti-Gbaguidi, Sophie Colin, Bart Staels. Nature Reviews Cardiology, 2015

632 citations recorded by the source corpus.

Subfields: Epidemiology, Immunology, Molecular Biology

View Paper: https://www.semanticscholar.org/paper/2ff9f1af3af3dd4c5a683bbb35da6052cd84beb1

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Epidemiology

1. I. Graham
2. Børge G. Nordestgaard
3. J. Bentzon
4. Jay D. Horton
5. Alberico L. Catapano

In Immunology

1. J. Bentzon
2. Alberico L. Catapano

In Molecular Biology

1. I. Graham
2. Jay D. Horton
3. Alberico L. Catapano
4. Ulrich Laufs

Citations in the corpus

• Macrophage Phenotype and Function in Different Stages of Atherosclerosis (2016, neutral): “These include the M(Hb) and Mhem populations,18 which are resistant to lipid loading and are induced by exposure to hemoglobin–haptoglobin complexes and haem in vitro, respectively; the Mox macrophage population, which is induced by exposure to oxidized phospholipids in vitro and is characterized by”
• Low-density lipoproteins cause atherosclerotic cardiovascular disease: pathophysiological, genetic, and therapeutic insights: a consensus statement from the European Atherosclerosis Society Consensus Panel (2020, background): “allowing macrophage subsets to participate in almost every stage of atherosclerosis.(138) Several DAMPs, generated by modification of retained LDL, induce”
• The Reactive Oxygen Species in Macrophage Polarization: Reflecting Its Dual Role in Progression and Treatment of Human Diseases (2016, background): “The involvement of M1/M2 in obesity, atherosclerosis, and cancer is briefly discussed below; the comprehensive reviews on human diseases have been published elsewhere [28–30].”