T cell– and B cell–independent adaptive immunity mediated by natural killer cells
By J. O’Leary, Mahmoud Goodarzi, Danielle L. Drayton, Ulrich H. von Andrian. Nature Immunology, 2006
896 citations recorded by the source corpus.
Subfields: Immunology
View Paper: https://www.semanticscholar.org/paper/2a6796d70ad07f97f2e908382ab4bf57d1f59af0
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Citations in the corpus
- Targeting natural killer cells and natural killer T cells in cancer (2012, background): “In addition, hapten- and virus-specific memory NK cells have been describe”
- Functions of natural killer cells (2008, background): “It has been recently reported that NK cells can mediate hapten-specific recall responses, independent of B cells and T cells, in a model of contact hypersensitivit”
- Dendritic cells prime natural killer cells by trans-presenting interleukin 15. (2007, background): “, 2005), and a recent report indicating that NK cells are capable of immunological ‘‘memory’’ (O’Leary et al., 2006), NK cells appear to be much more related to lymphocytes of the adaptive immune system than previously appreciated.”
- Candida albicans Infection Affords Protection against Reinfection via Functional Reprogramming of Monocytes (2012, background): “Recently, NK cells, another prototypical type of innate immune cells, have been demonstrated to possess memory characteristics (O’Leary et al., 2006; Paust et al., 2010; Sun et al., 2009).”
- Trained immunity: a program of innate immune memory in health and disease (2016, background): “, IL-12, IL-15, and IL-18) (32) or hapten sensitization, which induced long-lived NK cells that mediate contact hypersensitivity and long-lived antigen-specific recall responses independently of B and T cells (30).”
- Natural Killer Cells: Development, Maturation, and Clinical Utilization (2018, background): “However, the separation of these cells with regards to their contributions to adaptive immunity has recently been reconsidered due to the discovery of NK cells that exhibit immunological memory (160, 221).”
- Bacille Calmette-Guérin induces NOD2-dependent nonspecific protection from reinfection via epigenetic reprogramming of monocytes (2012, background): “Indeed,murineCMV infection can induce protection from reinfection in aT/B-cell–independent and natural killer cell–dependent fashion (4, 32), and systemic infection with C.”